Molecular participants in mitochondrial cell death channel formation during neuronal ischemia

Experimental Neurology

Volumn 218, Issue 2, 2009, Pages 203-212

  Jonas, Elizabeth Ann ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; BCL 2; Brain ischemia; Ion channel; Mitochondria; Mitochondrial permeability transition; Programmed cell death; Synaptic transmission; VDAC

Indexed keywords

CALCIUM; CYTOCHROME C; ION CHANNEL; POTASSIUM; PROTEIN BAD; PROTEIN BCL 2; PROTEIN BCL XL; SODIUM; VOLTAGE DEPENDENT ANION CHANNEL; VOLTAGE DEPENDENT ANION CHANNEL 2; ZINC;

BRAIN ISCHEMIA; CALCIUM TRANSPORT; CELL COMPARTMENTALIZATION; CELL DEATH; CELL HYPOXIA; CELL MEMBRANE PERMEABILITY; CELL MEMBRANE POTENTIAL; COMPLEX FORMATION; EXCITOTOXICITY; HIPPOCAMPUS; HUMAN; ION CONDUCTANCE; MITOCHONDRIAL PERMEABILITY; MITOCHONDRIAL RESPIRATION; MITOCHONDRION; MOLECULAR INTERACTION; NERVE CELL NECROSIS; NERVE CELL PLASTICITY; NEUROPATHOLOGY; NONHUMAN; POTASSIUM TRANSPORT; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN FUNCTION; REGULATORY MECHANISM; REVIEW; SIGNAL TRANSDUCTION; SODIUM TRANSPORT; SYNAPTIC TRANSMISSION;

ANIMALS; APOPTOSIS; CELL MEMBRANE PERMEABILITY; HUMANS; ION CHANNELS; ISCHEMIA; MITOCHONDRIA; MITOCHONDRIAL MEMBRANES; NEURONS; PROTO-ONCOGENE PROTEINS C-BCL-2; VOLTAGE-DEPENDENT ANION CHANNELS;

EID: 67649813198     PISSN: 00144886     EISSN: 10902430     Source Type: Journal    
DOI: 10.1016/j.expneurol.2009.03.025     Document Type: Review

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