H2AX is required for cell cycle arrest via the p53/p21 pathway

Molecular and Cellular Biology

Volumn 29, Issue 10, 2009, Pages 2828-2840

  Fragkos, Michalis ^a   Jurvansuu, Jaana ^a,b   Beard, Peter ^a  

^a EPFL   (Switzerland)

^b UNIVERSITY OF HELSINKI   (Finland)

Author keywords

[No Author keywords available]

Indexed keywords

ATM PROTEIN; ATR PROTEIN; CASPASE; HISTONE H2AX; PROTEIN P21; PROTEIN P53; ATR PROTEIN, HUMAN; CELL CYCLE PROTEIN; CYCLIN DEPENDENT KINASE INHIBITOR 1A; DNA BINDING PROTEIN; H2AFX PROTEIN, HUMAN; HISTONE; PROTEIN SERINE THREONINE KINASE; TUMOR SUPPRESSOR PROTEIN;

ADENOVIRUS; ARTICLE; CELL CYCLE ARREST; CONTROLLED STUDY; DIFFUSION; DNA DAMAGE; DNA REPAIR; ENZYME ACTIVATION; ENZYME INHIBITION; GENOME; HUMAN; HUMAN CELL; MITOSIS; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; RNA INTERFERENCE; SIGNAL TRANSDUCTION; VIRUS INDUCTION; VIRUS INFECTION; VIRUS REPLICATION; ADENO ASSOCIATED VIRUS; CELL CYCLE; CELL LINE; CELL NUCLEUS; GENETICS; METABOLISM; PHYSIOLOGY;

ADENO-ASSOCIATED VIRUS;

CELL CYCLE; CELL CYCLE PROTEINS; CELL LINE; CELL NUCLEUS; CYCLIN-DEPENDENT KINASE INHIBITOR P21; DEPENDOVIRUS; DNA DAMAGE; DNA REPAIR; DNA-BINDING PROTEINS; HISTONES; HUMANS; PROTEIN-SERINE-THREONINE KINASES; RNA INTERFERENCE; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 66349084196     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.01830-08     Document Type: Article

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