Mitochondrial perturbation attenuates bile acid-induced cytotoxicity

Cell Biology and Toxicology

Volumn 21, Issue 5-6, 2005, Pages 215-231

  Payne, C M ^a,b,d   Crowley Weber, C L ^a   Dvorak, K ^a,b   Bernstein, C ^a   Bernstein, H ^a,b   Holubec, H ^a   Crowley, C ^a   Garewal, H ^a,b,c  

^a UNIVERSITY OF ARIZONA   (United States)

^b UNIVERSITY OF ARIZONA   (United States)

^c SOUTHERN ARIZONA VA HEALTH CARE SYSTEM   (United States)

^d UNIVERSITY OF ARIZONA COLLEGE OF MEDICINE   (United States)

Author keywords

Apoptosis; Deoxycholate; ER stress; Mitochondria; Nucleolus

Indexed keywords

ANTIMYCIN A1; BILE ACID; DEOXYCHOLIC ACID; MITOCHONDRIAL ENZYME; MYXOTHIAZOL; OLIGOMYCIN; ROTENONE; THENOYLTRIFLUOROACETONE;

APOPTOSIS; ARTICLE; CONTROLLED STUDY; CYTOTOXICITY; ENDOPLASMIC RETICULUM; ENZYME INHIBITION; HUMAN; HUMAN CELL; MITOCHONDRION; NUCLEOLUS; OXIDATIVE STRESS; POLYMERIZATION; PRIORITY JOURNAL;

ANTIMYCIN A; APOPTOSIS; BILE ACIDS AND SALTS; CELL NUCLEOLUS; DEOXYCHOLIC ACID; ELECTRON TRANSPORT COMPLEX III; ENDOPLASMIC RETICULUM; HCT116 CELLS; HUMANS; MEMBRANE POTENTIALS; METHACRYLATES; MICROSCOPY, ELECTRON, TRANSMISSION; MITOCHONDRIA; MITOCHONDRIAL PROTON-TRANSLOCATING ATPASES; MITOCHONDRIAL SWELLING; OLIGOMYCINS; THIAZOLES; TIME FACTORS; UNCOUPLING AGENTS;

EID: 28444448048     PISSN: 07422091     EISSN: None     Source Type: Journal    
DOI: 10.1007/s10565-005-0166-6     Document Type: Article

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