Roles of CHOP/GADD153 in endoplasmic reticulum stress

Cell Death and Differentiation

Volumn 11, Issue 4, 2004, Pages 381-389

  Oyadomari, S ^a,b   Mori, M ^a  

^a KUMAMOTO UNIVERSITY   (Japan)

^b NEW YORK UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; Diabetes; Endoplasmic reticulum; Nitric oxide; Pancreatic cells; Transcription factor

Indexed keywords

CHAPERONE; GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153;

BRAIN ISCHEMIA; CELL DIFFERENTIATION; CELL GROWTH; DEGENERATIVE DISEASE; DIABETES MELLITUS; ENDOPLASMIC RETICULUM; HOMEOSTASIS; HUMAN; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN FOLDING; PROTEIN FUNCTION; PROTEIN STRUCTURE; PROTEIN SYNTHESIS; QUALITY CONTROL; REVIEW; SIGNAL TRANSDUCTION; STRESS; TRANSCRIPTION REGULATION; TRANSLATION REGULATION; UPREGULATION;

ANIMALS; APOPTOSIS; CCAAT-ENHANCER-BINDING PROTEINS; CELL DIFFERENTIATION; CELL DIVISION; DISEASE; ENDOPLASMIC RETICULUM; HUMANS; MICE; STRESS; TRANSCRIPTION FACTOR CHOP; TRANSCRIPTION FACTORS;

EID: 1842843855     PISSN: 13509047     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.cdd.4401373     Document Type: Review

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