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Volumn 98, Issue 19, 2001, Pages 10845-10850
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Nitric oxide-induced apoptosis in pancreatic β cells is mediated by the endoplasmic reticulum stress pathway
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Author keywords
[No Author keywords available]
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Indexed keywords
CALCIUM;
CALRETICULIN;
CCAAT ENHANCER BINDING PROTEIN;
DNA;
GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153;
N ACETYL S NITROSOPENICILLAMINE;
NITRIC OXIDE;
NITRIC OXIDE DONOR;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
APOPTOSIS;
ARTICLE;
CELL DEATH;
CELL GROWTH;
CONTROLLED STUDY;
DNA DAMAGE;
ENDOPLASMIC RETICULUM;
FEMALE;
INSULIN DEPENDENT DIABETES MELLITUS;
KNOCKOUT MOUSE;
MALE;
MOUSE;
NONHUMAN;
NUCLEOTIDE SEQUENCE;
OXIDATIVE STRESS;
PANCREAS ISLET BETA CELL;
PATHOGENESIS;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
ANIMALS;
APOPTOSIS;
CALCIUM;
CALCIUM-BINDING PROTEINS;
CALRETICULIN;
CCAAT-ENHANCER-BINDING PROTEINS;
CYCLIC GMP;
DNA DAMAGE;
DRUG RESISTANCE;
ENDOPLASMIC RETICULUM;
HOMEOSTASIS;
INTERFERON TYPE II;
INTERLEUKIN-1;
ISLETS OF LANGERHANS;
MICE;
NITRIC OXIDE;
NITRIC OXIDE DONORS;
PENICILLAMINE;
RIBONUCLEOPROTEINS;
TRANSCRIPTION FACTOR CHOP;
TRANSCRIPTION FACTORS;
TUMOR CELLS, CULTURED;
TUMOR NECROSIS FACTOR-ALPHA;
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EID: 0035845568
PISSN: 00278424
EISSN: None
Source Type: Journal
DOI: 10.1073/pnas.191207498 Document Type: Article |
Times cited : (526)
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References (42)
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