-
1
-
-
0035856952
-
β-Cell death during progression to diabetes
-
Mathis D, Vence L, Benoist C. β-Cell death during progression to diabetes. Nature 414: 792-798, 2001.
-
(2001)
Nature
, vol.414
, pp. 792-798
-
-
Mathis, D.1
Vence, L.2
Benoist, C.3
-
2
-
-
0035668779
-
A choice of death -the signal-transduction of immune-mediated β-cell apoptosis
-
Eizirik DL, Mandrup-Poulsen T. A choice of death -the signal-transduction of immune-mediated β-cell apoptosis. Diabetologia 44: 2115-2133, 2001.
-
(2001)
Diabetologia
, vol.44
, pp. 2115-2133
-
-
Eizirik, D.L.1
Mandrup-Poulsen, T.2
-
3
-
-
0025777354
-
Heat shock, stress proteins, chaperones, and proteotoxicity
-
Hightower LE. Heat shock, stress proteins, chaperones, and proteotoxicity. Cell 66: 191-197, 1991.
-
(1991)
Cell
, vol.66
, pp. 191-197
-
-
Hightower, L.E.1
-
4
-
-
0034578389
-
Aggresomes, inclusion bodies and protein aggregation
-
Kopito RR. Aggresomes, inclusion bodies and protein aggregation. Trends Cell Biol 10: 524-530, 2000.
-
(2000)
Trends Cell Biol
, vol.10
, pp. 524-530
-
-
Kopito, R.R.1
-
5
-
-
0034716887
-
Tripartite management of unfolded proteins in the endoplasmic reticulum
-
Mori K. Tripartite management of unfolded proteins in the endoplasmic reticulum. Cell 101: 451-454, 2000.
-
(2000)
Cell
, vol.101
, pp. 451-454
-
-
Mori, K.1
-
6
-
-
0036086064
-
The unfolded protein response in nutrient sensing and differentiation
-
Kaufman RJ, Scheuner D, Schroder M, et al. The unfolded protein response in nutrient sensing and differentiation. Nat Rev Mol Cell Biol 3: 411-421, 2002.
-
(2002)
Nat. Rev. Mol. Cell Biol.
, vol.3
, pp. 411-421
-
-
Kaufman, R.J.1
Scheuner, D.2
Schroder, M.3
-
7
-
-
0036022403
-
Endoplasmic reticulum stress-mediated apoptosis in pancreatic β-cells
-
Oyadomari S, Araki E, Mori M. Endoplasmic reticulum stress-mediated apoptosis in pancreatic β-cells. Apoptosis 7: 335-345, 2002.
-
(2002)
Apoptosis
, vol.7
, pp. 335-345
-
-
Oyadomari, S.1
Araki, E.2
Mori, M.3
-
8
-
-
0026546365
-
CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription
-
Ron D, Habener JF. CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription. Genes Dev 6: 439-453, 1992.
-
(1992)
Genes Dev.
, vol.6
, pp. 439-453
-
-
Ron, D.1
Habener, J.F.2
-
9
-
-
8944259440
-
Signals from the stressed endoplasmic reticulum induce C/EBP-homologous protein
-
(CHOP/GADD153)
-
Wang XZ, Lawson B, Brewer JW, et al. Signals from the stressed endoplasmic reticulum induce C/EBP-homologous protein (CHOP/GADD153). Mol Cell Biol 16: 4273-4280, 1996.
-
(1996)
Mol. Cell Biol.
, vol.16
, pp. 4273-4280
-
-
Wang, X.Z.1
Lawson, B.2
Brewer, J.W.3
-
10
-
-
23444443039
-
CHOP (GADD153) and its oncogenic variant, TLS-CHOP, have opposing effects on the induction of G1/S arrest
-
Barone MV, Crozat A, Tabaee A, Philipson L, Ron D. CHOP (GADD153) and its oncogenic variant, TLS-CHOP, have opposing effects on the induction of G1/S arrest. Genes Dev 8: 453-464, 1994.
-
(1994)
Genes Dev.
, vol.8
, pp. 453-464
-
-
Barone, M.V.1
Crozat, A.2
Tabaee, A.3
Philipson, L.4
Ron, D.5
-
11
-
-
0030597160
-
Ectopic expression of CHOP (GADD153) induces apoptosis in M1 myeloblastic leukemia cells
-
Matsumoto M, Minami M, Takeda K, Sakao Y, Akira S. Ectopic expression of CHOP (GADD153) induces apoptosis in M1 myeloblastic leukemia cells. FEBS Lett 395: 143-147, 1996.
-
(1996)
FEBS Lett.
, vol.395
, pp. 143-147
-
-
Matsumoto, M.1
Minami, M.2
Takeda, K.3
Sakao, Y.4
Akira, S.5
-
12
-
-
0032054744
-
CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum
-
Zinszner H, Kuroda M, Wang X, et al. CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum. Genes Dev 12: 982-995, 1998.
-
(1998)
Genes Dev.
, vol.12
, pp. 982-995
-
-
Zinszner, H.1
Kuroda, M.2
Wang, X.3
-
13
-
-
0035845568
-
Nitric oxide-induced apoptosis in pancreatic β cells is mediated by the endoplasmic reticulum stress pathway
-
Oyadomari S, Takeda K, Takiguchi M, et al. Nitric oxide-induced apoptosis in pancreatic β cells is mediated by the endoplasmic reticulum stress pathway. Proc Natl Acad Sci USA 98: 10845-10850, 2001.
-
(2001)
Proc. Natl. Acad. Sci. USA
, vol.98
, pp. 10845-10850
-
-
Oyadomari, S.1
Takeda, K.2
Takiguchi, M.3
-
14
-
-
0036175128
-
Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes
-
Oyadomari S, Koizumi A, Takeda K, et al. Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes. J Clin Invest 109: 525-532, 2002.
-
(2002)
J. Clin. Invest.
, vol.109
, pp. 525-532
-
-
Oyadomari, S.1
Koizumi, A.2
Takeda, K.3
-
15
-
-
0032190546
-
Cloning of mammalian Ire1 reveals diversity in the ER stress responses
-
Wang XZ, Harding HP, Zhang Y, Jolicoeur EM, Kuroda M, Ron D. Cloning of mammalian Ire1 reveals diversity in the ER stress responses. EMBO J 17: 5708-5717, 1998.
-
(1998)
EMBO J.
, vol.17
, pp. 5708-5717
-
-
Wang, X.Z.1
Harding, H.P.2
Zhang, Y.3
Jolicoeur, E.M.4
Kuroda, M.5
Ron, D.6
-
16
-
-
0033634654
-
Regulated translation initiation controls stress-induced gene expression in mammalian cells
-
Harding HP, Novoa I, Zhang Y, et al. Regulated translation initiation controls stress-induced gene expression in mammalian cells. Mol Cell 6: 1099-1108, 2000.
-
(2000)
Mol. Cell
, vol.6
, pp. 1099-1108
-
-
Harding, H.P.1
Novoa, I.2
Zhang, Y.3
-
17
-
-
0033815971
-
ATF6 activated by proteolysis binds in the presence of NF-Y (CBF) directly to the cis-acting element responsible for the mammalian unfolded protein response
-
Yoshida H, Okada T, Haze K, et al. ATF6 activated by proteolysis binds in the presence of NF-Y (CBF) directly to the cis-acting element responsible for the mammalian unfolded protein response. Mol Cell Biol 20: 6755-6767, 2000.
-
(2000)
Mol. Cell Biol.
, vol.20
, pp. 6755-6767
-
-
Yoshida, H.1
Okada, T.2
Haze, K.3
-
18
-
-
0032940099
-
CHOP-Dependent stress-inducible expression of a novel form of carbonic anhydrase VI
-
Sok J, Wang XZ, Batchvarova N, Kuroda M, Harding H, Ron D. CHOP-Dependent stress-inducible expression of a novel form of carbonic anhydrase VI. Mol Cell Biol 19: 495-504, 1999.
-
(1999)
Mol. Cell Biol.
, vol.19
, pp. 495-504
-
-
Sok, J.1
Wang, X.Z.2
Batchvarova, N.3
Kuroda, M.4
Harding, H.5
Ron, D.6
-
19
-
-
9844257587
-
Inhibition of Bax channel-forming activity by Bcl-2
-
Antonsson B, Conti F, Ciavatta A, et al. Inhibition of Bax channel-forming activity by Bcl-2. Science 277: 370-372, 1997.
-
(1997)
Science
, vol.277
, pp. 370-372
-
-
Antonsson, B.1
Conti, F.2
Ciavatta, A.3
-
20
-
-
0035144493
-
Gadd153 sensitizes cells to endoplasmic reticulum stress by down-regulating Bcl2 and perturbing the cellular redox state
-
McCullough KD, Martindale JL, Klotz LO, Aw TY, Holbrook NJ. Gadd153 sensitizes cells to endoplasmic reticulum stress by down-regulating Bcl2 and perturbing the cellular redox state. Mol Cell Biol 21: 1249-1259, 2001.
-
(2001)
Mol. Cell Biol.
, vol.21
, pp. 1249-1259
-
-
McCullough, K.D.1
Martindale, J.L.2
Klotz, L.O.3
Aw, T.Y.4
Holbrook, N.J.5
-
21
-
-
0034723235
-
Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1
-
Urano F, Wang X, Bertolotti A, et al. Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1. Science 287: 664-666, 2000.
-
(2000)
Science
, vol.287
, pp. 664-666
-
-
Urano, F.1
Wang, X.2
Bertolotti, A.3
-
22
-
-
0036606540
-
ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats
-
Nishitoh H, Matsuzawa A, Tobiume K, et al. ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats. Genes Dev 16: 1345-1355, 2002.
-
(2002)
Genes Dev.
, vol.16
, pp. 1345-1355
-
-
Nishitoh, H.1
Matsuzawa, A.2
Tobiume, K.3
-
23
-
-
0034610743
-
Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta
-
Nakagawa T, Zhu H, Morishima N, et al. Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta. Nature 403: 98-103, 2000.
-
(2000)
Nature
, vol.403
, pp. 98-103
-
-
Nakagawa, T.1
Zhu, H.2
Morishima, N.3
-
24
-
-
0034698878
-
Cross-talk between two cysteine protease families. Activation of caspase-12 by calpain in apoptosis
-
Nakagawa T, Yuan J. Cross-talk between two cysteine protease families. Activation of caspase-12 by calpain in apoptosis. J Cell Biol 150: 887-894, 2000.
-
(2000)
J. Cell Biol.
, vol.150
, pp. 887-894
-
-
Nakagawa, T.1
Yuan, J.2
-
25
-
-
0035957929
-
Activation of caspase-12, an endoplastic reticulum (ER) resident caspase, through tumor necrosis factor receptor-associated factor 2-dependent mechanism in response to the ER stress
-
Yoneda T, Imaizumi K, Oono K, et al. Activation of caspase-12, an endoplastic reticulum (ER) resident caspase, through tumor necrosis factor receptor-associated factor 2-dependent mechanism in response to the ER stress. J Biol Chem 276: 13935-13940, 2001.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 13935-13940
-
-
Yoneda, T.1
Imaizumi, K.2
Oono, K.3
-
26
-
-
0035823579
-
Coupling endoplasmic reticulum stress to the cell death program. Mechanism of caspase activation
-
Rao RV, Hermel E, Castro-Obregon S, et al. Coupling endoplasmic reticulum stress to the cell death program. Mechanism of caspase activation. J Biol Chem 276: 33869-33874, 2001.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 33869-33874
-
-
Rao, R.V.1
Hermel, E.2
Castro-Obregon, S.3
-
27
-
-
0036294646
-
Human caspase 12 has acquired deleterious mutations
-
Fischer H, Koenig U, Eckhart L, Tschachler E. Human caspase 12 has acquired deleterious mutations. Biochem Biophys Res Commun 293: 722-726, 2002.
-
(2002)
Biochem. Biophys. Res. Commun.
, vol.293
, pp. 722-726
-
-
Fischer, H.1
Koenig, U.2
Eckhart, L.3
Tschachler, E.4
-
28
-
-
0030907296
-
A novel locus, Mody4, distal to D7Mit189 on chromosome 7 determines early-onset NIDDM in nonobese C57BL/6 (Akita) mutant mice
-
Yoshioka M, Kayo T, Ikeda T, Koizumi A. A novel locus, Mody4, distal to D7Mit189 on chromosome 7 determines early-onset NIDDM in nonobese C57BL/6 (Akita) mutant mice. Diabetes 46: 887-894, 1997.
-
(1997)
Diabetes
, vol.46
, pp. 887-894
-
-
Yoshioka, M.1
Kayo, T.2
Ikeda, T.3
Koizumi, A.4
-
29
-
-
0032524013
-
Mapping of murine diabetogenic gene mody on chromosome 7 at D7Mit258 and its involvement in pancreatic islet and β cell development during the perinatal period
-
Kayo T, Koizumi A. Mapping of murine diabetogenic gene mody on chromosome 7 at D7Mit258 and its involvement in pancreatic islet and β cell development during the perinatal period. J Clin Invest 101: 2112-2118, 1998.
-
(1998)
J. Clin. Invest.
, vol.101
, pp. 2112-2118
-
-
Kayo, T.1
Koizumi, A.2
-
30
-
-
0032918044
-
A mutation in the insulin 2 gene induces diabetes with severe pancreatic beta-cell dysfunction in the Mody mouse
-
Wang J, Takeuchi T, Tanaka S, et al. A mutation in the insulin 2 gene induces diabetes with severe pancreatic beta-cell dysfunction in the Mody mouse. J Clin Invest 103: 27-37, 1999.
-
(1999)
J. Clin. Invest.
, vol.103
, pp. 27-37
-
-
Wang, J.1
Takeuchi, T.2
Tanaka, S.3
-
31
-
-
0035129172
-
Compensatory responses in mice carrying a null mutation for Ins1 or Ins2
-
Leroux L, Desbois P, Lamotte L, et al. Compensatory responses in mice carrying a null mutation for Ins1 or Ins2. Diabetes 50 Suppl 1: S150-153, 2001.
-
(2001)
Diabetes
, vol.50
, Issue.SUPPL. 1
-
-
Leroux, L.1
Desbois, P.2
Lamotte, L.3
-
32
-
-
0034968330
-
Diabetes mellitus and exocrine pancreatic dysfunction in perk-/-mice reveals a role for translational control in secretory cell survival
-
Harding HP, Zeng H, Zhang Y, et al. Diabetes mellitus and exocrine pancreatic dysfunction in perk-/-mice reveals a role for translational control in secretory cell survival. Mol Cell 7: 1153-1163, 2001.
-
(2001)
Mol. Cell
, vol.7
, pp. 1153-1163
-
-
Harding, H.P.1
Zeng, H.2
Zhang, Y.3
-
33
-
-
0036091476
-
The PERK eukaryotic initiation factor 2α kinase is required for the development of the skeletal system, postnatal growth, and the function and viability of the pancreas
-
Zhang P, McGrath B, Li S, et al. The PERK eukaryotic initiation factor 2α kinase is required for the development of the skeletal system, postnatal growth, and the function and viability of the pancreas. Mol Cell Biol 22: 3864-3874, 2002.
-
(2002)
Mol. Cell Biol.
, vol.22
, pp. 3864-3874
-
-
Zhang, P.1
McGrath, B.2
Li, S.3
-
34
-
-
0034425698
-
EIF2AK3, encoding translation initiation factor 2-α kinase 3, is mutated in patients with Wolcott-Rallison syndrome
-
Delepine M, Nicolino M, Barrett T, Golamaully M, Lathrop GM, Julier C. EIF2AK3, encoding translation initiation factor 2-α kinase 3, is mutated in patients with Wolcott-Rallison syndrome. Nat Genet 25: 406-409, 2000.
-
(2000)
Nat. Genet.
, vol.25
, pp. 406-409
-
-
Delepine, M.1
Nicolino, M.2
Barrett, T.3
Golamaully, M.4
Lathrop, G.M.5
Julier, C.6
-
36
-
-
0034973982
-
Translational control is required for the unfolded protein response and in vivo glucose homeostasis
-
Scheuner D, Song B, McEwen E, et al. Translational control is required for the unfolded protein response and in vivo glucose homeostasis. Mol Cell 7: 1165-1176, 2001.
-
(2001)
Mol. Cell
, vol.7
, pp. 1165-1176
-
-
Scheuner, D.1
Song, B.2
McEwen, E.3
-
37
-
-
0034066505
-
Effect of nitric oxide on endoplasmic reticulum calcium homeostasis, protein synthesis and energy metabolism
-
Doutheil J, Althausen S, Treiman M, Paschen W. Effect of nitric oxide on endoplasmic reticulum calcium homeostasis, protein synthesis and energy metabolism. Cell Calcium 27: 107-115, 2000.
-
(2000)
Cell Calcium
, vol.27
, pp. 107-115
-
-
Doutheil, J.1
Althausen, S.2
Treiman, M.3
Paschen, W.4
-
38
-
-
0034235397
-
Calcium, a signaling molecule in the endoplasmic reticulum?
-
Corbett EF, Michalak M. Calcium, a signaling molecule in the endoplasmic reticulum? Trends Biochem Sci 25: 307-311, 2000.
-
(2000)
Trends Biochem. Sci.
, vol.25
, pp. 307-311
-
-
Corbett, E.F.1
Michalak, M.2
-
39
-
-
0035029867
-
Identification of novel cytokine-induced genes in pancreatic β-cells by high-density oligonucleotide arrays
-
Cardozo AK, Kruhoffer M, Leeman R, Orntoft T, Eizirik DL. Identification of novel cytokine-induced genes in pancreatic β-cells by high-density oligonucleotide arrays. Diabetes 50: 909-920, 2001.
-
(2001)
Diabetes
, vol.50
, pp. 909-920
-
-
Cardozo, A.K.1
Kruhoffer, M.2
Leeman, R.3
Orntoft, T.4
Eizirik, D.L.5
-
40
-
-
0035812912
-
Induction of CHOP and apoptosis by nitric oxide in p53-deficient microglial cells
-
Kawahara K, Oyadomari S, Gotoh T, Kohsaka S, Nakayama H, Mori M. Induction of CHOP and apoptosis by nitric oxide in p53-deficient microglial cells. FEBS Lett 506: 135-139, 2001.
-
(2001)
FEBS Lett.
, vol.506
, pp. 135-139
-
-
Kawahara, K.1
Oyadomari, S.2
Gotoh, T.3
Kohsaka, S.4
Nakayama, H.5
Mori, M.6
-
41
-
-
0037023726
-
Nitric oxide-induced apoptosis in RAW 264.7 macrophages is mediated by endoplasmic reticulum stress pathway involving ATF6 and CHOP
-
Gotoh T, Oyadomari S, Mori K, Mori M. Nitric oxide-induced apoptosis in RAW 264.7 macrophages is mediated by endoplasmic reticulum stress pathway involving ATF6 and CHOP. J Biol Chem 277: 12343-12350, 2002.
-
(2002)
J. Biol. Chem.
, vol.277
, pp. 12343-12350
-
-
Gotoh, T.1
Oyadomari, S.2
Mori, K.3
Mori, M.4
-
42
-
-
0035969513
-
Islet amyloid and type 2 diabetes: From molecular misfolding to islet pathophysiology
-
1537
-
Jaikaran ET, Clark A. Islet amyloid and type 2 diabetes: from molecular misfolding to islet pathophysiology. Biochim Biophys Acta 1537: 179-203, 2001.
-
(2001)
Biochim. Biophys. Acta
, pp. 179-203
-
-
Jaikaran, E.T.1
Clark, A.2
-
43
-
-
0029818742
-
Missense mutation of amylin gene (S20G) in Japanese NIDDM patients
-
Sakagashira S, Sanke T, Hanabusa T, et al. Missense mutation of amylin gene (S20G) in Japanese NIDDM patients. Diabetes 45: 1279-1281, 1996.
-
(1996)
Diabetes
, vol.45
, pp. 1279-1281
-
-
Sakagashira, S.1
Sanke, T.2
Hanabusa, T.3
-
44
-
-
0034126660
-
Islet amyloid polypeptide in the islets of Langerhans: Friend or foe?
-
Gebre-Medhin S, Olofsson C, Mulder H. Islet amyloid polypeptide in the islets of Langerhans: friend or foe? Diabetologia 43: 687-695, 2000.
-
(2000)
Diabetologia
, vol.43
, pp. 687-695
-
-
Gebre-Medhin, S.1
Olofsson, C.2
Mulder, H.3
-
45
-
-
0037041420
-
Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases
-
Bucciantini M, Giannoni E, Chiti F, et al. Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases. Nature 416: 507-511, 2002.
-
(2002)
Nature
, vol.416
, pp. 507-511
-
-
Bucciantini, M.1
Giannoni, E.2
Chiti, F.3
-
46
-
-
0028303844
-
Pancreatic islet cell toxicity of amylin associated with type-2 diabetes mellitus
-
Lorenzo A, Razzaboni B, Weir GC, Yankner BA. Pancreatic islet cell toxicity of amylin associated with type-2 diabetes mellitus. Nature 368: 756-760, 1994.
-
(1994)
Nature
, vol.368
, pp. 756-760
-
-
Lorenzo, A.1
Razzaboni, B.2
Weir, G.C.3
Yankner, B.A.4
-
47
-
-
0029901739
-
Spontaneous diabetes mellitus in transgenic mice expressing human islet amyloid polypeptide
-
Janson J, Soeller WC, Roche PC, et al. Spontaneous diabetes mellitus in transgenic mice expressing human islet amyloid polypeptide. Proc Natl Acad Sci USA 93: 7283-7288, 1996.
-
(1996)
Proc. Natl. Acad. Sci. USA
, vol.93
, pp. 7283-7288
-
-
Janson, J.1
Soeller, W.C.2
Roche, P.C.3
-
48
-
-
0027451036
-
Chronic overproduction of islet amyloid polypeptide/amylin in transgenic mice: Lysosomal localization of human islet amyloid polypeptide and lack of marked hyperglycaemia or hyperinsulinaemia
-
Hoppener JW, Verbeek JS, de Koning EJ, et al. Chronic overproduction of islet amyloid polypeptide/amylin in transgenic mice: lysosomal localization of human islet amyloid polypeptide and lack of marked hyperglycaemia or hyperinsulinaemia. Diabetologia 36: 1258-1265, 1993.
-
(1993)
Diabetologia
, vol.36
, pp. 1258-1265
-
-
Hoppener, J.W.1
Verbeek, J.S.2
de Koning, E.J.3
-
49
-
-
0028598171
-
Pancreatic expression and secretion of human islet amyloid polypeptide in a transgenic mouse
-
D'Alessio DA, Verchere CB, Kahn SE, et al. Pancreatic expression and secretion of human islet amyloid polypeptide in a transgenic mouse. Diabetes 43: 1457-1461, 1994.
-
(1994)
Diabetes
, vol.43
, pp. 1457-1461
-
-
D'Alessio, D.A.1
Verchere, C.B.2
Kahn, S.E.3
-
50
-
-
0141780910
-
Extensive islet amyloid formation is induced by development of Type II diabetes mellitus and contributes to its progression: Pathogenesis of diabetes in a mouse model
-
Hoppener JW, Oosterwijk C, Nieuwenhuis MG, et al. Extensive islet amyloid formation is induced by development of Type II diabetes mellitus and contributes to its progression: pathogenesis of diabetes in a mouse model. Diabetologia 42: 427-434, 1999.
-
(1999)
Diabetologia
, vol.42
, pp. 427-434
-
-
Hoppener, J.W.1
Oosterwijk, C.2
Nieuwenhuis, M.G.3
-
51
-
-
0029978228
-
Islet amyloid formation associated with hyperglycemia in transgenic mice with pancreatic β cell expression of human islet amyloid polypeptide
-
Verchere CB, D'Alessio DA, Palmiter RD, et al. Islet amyloid formation associated with hyperglycemia in transgenic mice with pancreatic β cell expression of human islet amyloid polypeptide. Proc Natl Acad Sci USA 93: 3492-3496, 1996.
-
(1996)
Proc. Natl. Acad. Sci. USA
, vol.93
, pp. 3492-3496
-
-
Verchere, C.B.1
D'Alessio, D.A.2
Palmiter, R.D.3
|