Human hyper-IgE syndrome: singular or plural?

Mammalian Genome

Volumn 29, Issue 7-8, 2018, Pages 603-617

  Zhang, Qian ^a   Boisson, Bertrand ^a,b,c   Béziat, Vivien ^b,c   Puel, Anne ^a,b,c   Casanova, Jean Laurent ^a,b,c,d,e  

^a ROCKEFELLER UNIVERSITY   (United States)

^b IMAGINE INSTITUTE   (France)

^c UNIVERSITÉ PARIS DESCARTES   (France)

^d HÔPITAL NECKER ENFANTS MALADES   (France)

^e HOWARD HUGHES MEDICAL INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

IMMUNOGLOBULIN E; STAT3 PROTEIN; BIOLOGICAL MARKER; DOCK8 PROTEIN, HUMAN; GUANINE NUCLEOTIDE EXCHANGE FACTOR; PGM3 PROTEIN, HUMAN; PHOSPHOGLUCOMUTASE;

AUTOSOMAL DOMINANT DISORDER; CELL LINEAGE; DOCK8 DEFICIENCY; HUMAN; HYPER IGE SYNDROME; IMMUNITY; IMMUNOGLOBULIN BLOOD LEVEL; INFECTION; NONHUMAN; PGM3 DEFICIENCY; PHENOTYPE; REVIEW; ANIMAL; ANTIBODY PRODUCTION; DEFICIENCY; DISEASE MODEL; DISEASE PREDISPOSITION; GENETIC PREDISPOSITION; GENETICS; IMMUNOLOGY; METABOLISM; SIGNAL TRANSDUCTION;

ANIMALS; ANTIBODY FORMATION; BIOMARKERS; DISEASE MODELS, ANIMAL; DISEASE SUSCEPTIBILITY; GENETIC PREDISPOSITION TO DISEASE; GUANINE NUCLEOTIDE EXCHANGE FACTORS; HUMANS; IMMUNOGLOBULIN E; JOB SYNDROME; PHENOTYPE; PHOSPHOGLUCOMUTASE; SIGNAL TRANSDUCTION; STAT3 TRANSCRIPTION FACTOR;

EID: 85051301835     PISSN: 09388990     EISSN: 14321777     Source Type: Journal    
DOI: 10.1007/s00335-018-9767-2     Document Type: Review

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