Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

Nature

Volumn 452, Issue 7188, 2008, Pages 773-776

  Milner, Joshua D ^a   Brenchley, Jason M ^a,c   Laurence, Arian ^b   Freeman, Alexandra F ^c   Hill, Brenna J ^a   Elias, Kevin M ^b,d   Kanno, Yuka ^b   Spalding, Christine ^c   Elloumi, Houda Z ^c   Paulson, Michelle L ^c   Davis, Joie ^c   Hsu, Amy ^c   Asher, Ava I ^a   O'Shea, John ^b   Holland, Steven M ^c   Paul, William E ^a   Douek, Daniel C ^a  

^a NATIONAL INSTITUTES OF HEALTH   (United States)

^b NATIONAL INSTITUTE OF ARTHRITIS AND MUSCULOSKELETAL AND SKIN DISEASES   (United States)

^c NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES   (United States)

^d VANDERBILT UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GAMMA INTERFERON; INTERLEUKIN 17; INTERLEUKIN 2; STAPHYLOCOCCUS ENTEROTOXIN B; STREPTOKINASE; TUMOR NECROSIS FACTOR;

ENZYME ACTIVITY; GENE EXPRESSION; MUTATION; RESPIRATORY DISEASE; SIGNALING; YEAST;

ADOLESCENT; ADULT; ARTICLE; BACTERIAL INFECTION; CANDIDA ALBICANS; CELL DIFFERENTIATION; CHILD; CLINICAL ARTICLE; CYTOKINE PRODUCTION; FEMALE; HELPER CELL; HUMAN; HYPER IGE SYNDROME; INFECTION SENSITIVITY; MALE; MYCOSIS; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; RECURRENT INFECTION; SIGNAL TRANSDUCTION; T LYMPHOCYTE;

BACTERIA (MICROORGANISMS); CANDIDA ALBICANS;

EID: 41449110468     PISSN: 00280836     EISSN: 14764687     Source Type: Journal    
DOI: 10.1038/nature06764     Document Type: Article

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