Blood glucose fluctuation accelerates renal injury involved to inhibit the AKT signaling pathway in diabetic rats

Endocrine

Volumn 53, Issue 1, 2016, Pages 81-96

  Ying, Changjiang ^a,b   Zhou, Xiaoyan ^b   Chang, Zhenzhen ^b   Ling, Hongwei ^b   Cheng, Xingbo ^a   Li, Wei ^b  

^a THE FIRST AFFILIATED HOSPITAL OF SOOCHOW UNIVERSITY   (China)

^b XUZHOU MEDICAL UNIVERSITY   (China)

Author keywords

Blood glucose fluctuation; Cleaved caspase 3; Diabetic nephropathy; p AKT

Indexed keywords

CASPASE 3; CREATININE; GLUCOSE; GLYCOGEN SYNTHASE KINASE 3BETA; HEMOGLOBIN A1C; INSULIN; MALONALDEHYDE; NITROGEN; PHOSPHOPROTEIN; PHOSPHOSERINE; PROTEIN BAX; PROTEIN BCL 2; PROTEIN KINASE B; STREPTOZOCIN; SUPEROXIDE DISMUTASE; GLUCOSE BLOOD LEVEL;

ADULT; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BODY WEIGHT; CONTROLLED STUDY; CREATININE BLOOD LEVEL; CREATININE CLEARANCE; DIABETIC NEPHROPATHY; ENZYME ACTIVITY; ENZYME INHIBITION; GLUCOSE BLOOD LEVEL; KIDNEY PARENCHYMA; KIDNEY STRUCTURE; LIPID DIET; MALE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; SIGNAL TRANSDUCTION; ANIMAL; EXPERIMENTAL DIABETES MELLITUS; KIDNEY; KIDNEY FUNCTION TEST; METABOLISM; PHOSPHORYLATION; PHYSIOLOGY; SPRAGUE DAWLEY RAT;

ANIMALS; BLOOD GLUCOSE; DIABETES MELLITUS, EXPERIMENTAL; DIABETIC NEPHROPATHIES; DIET, HIGH-FAT; KIDNEY; KIDNEY FUNCTION TESTS; MALE; PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS C-AKT; RATS; RATS, SPRAGUE-DAWLEY; SIGNAL TRANSDUCTION;

EID: 84957711607     PISSN: 1355008X     EISSN: 15590100     Source Type: Journal    
DOI: 10.1007/s12020-016-0867-z     Document Type: Article

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