Ischaemic conditioning strategies reduce ischaemia/reperfusion-induced organ injury

British Journal of Anaesthesia

Volumn 114, Issue 2, 2015, Pages 204-216

  Pac Soo, C K ^a,b   Mathew, H ^b   Ma, D ^a  

^a Department of Surgery and Cancer ^*  (United Kingdom)

^b WYCOMBE HOSPITAL   (United Kingdom)

Author keywords

cell signal pathway; ischaemia reperfusion injury; mitochondria; molecular mechanism; pre and post conditioning

Indexed keywords

ADENOSINE RECEPTOR; BRADYKININ RECEPTOR; CANNABINOID RECEPTOR; G PROTEIN COUPLED RECEPTOR; JANUS KINASE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; OPIATE RECEPTOR; PHOSPHATIDYLINOSITOL 3 KINASE; TUMOR NECROSIS FACTOR ALPHA;

AEROBIC METABOLISM; ANAEROBIC GLYCOLYSIS; APOPTOSIS; CELL DEATH; CELL PROTECTION; HUMAN; ISCHEMIC POSTCONDITIONING; ISCHEMIC PRECONDITIONING; LIPID PEROXIDATION; MEMBRANE TRANSPORT; MITOCHONDRIAL PERMEABILITY; MITOCHONDRION; ORGAN INJURY; PRIORITY JOURNAL; REPERFUSION INJURY; REVIEW; SIGNAL TRANSDUCTION; TISSUE PERFUSION; COMPLICATION; MYOCARDIAL REPERFUSION INJURY; PATHOPHYSIOLOGY; PROCEDURES;

HUMANS; ISCHEMIC POSTCONDITIONING; ISCHEMIC PRECONDITIONING; MYOCARDIAL REPERFUSION INJURY; REPERFUSION INJURY;

EID: 84922489573     PISSN: 00070912     EISSN: 14716771     Source Type: Journal    
DOI: 10.1093/bja/aeu302     Document Type: Review

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