Regulating RISK: A role for JAK-STAT signaling in postconditioning?

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 295, Issue 4, 2008, Pages

  Goodman, Michael D ^a,b   Koch, Sheryl E ^a   Fuller Bicer, Geraldine A ^a   Butler, Karyn L ^a  

^a UNIVERSITY OF CINCINNATI   (United States)

^b UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

Ischemia reperfusion; Phosphatidylinositol 3 kinase Akt; STAT3

Indexed keywords

2 MORPHOLINO 8 PHENYLCHROMONE; JANUS KINASE; JANUS KINASE 2; JANUS KINASE INHIBITOR; N BENZYL 2 CYANO 3 (3,4 DIHYDROXYPHENYL)ACRYLAMIDE; PHOSPHATIDYLINOSITOL 3 KINASE; PHOSPHATIDYLINOSITOL 3 KINASE INHIBITOR; PROTEIN KINASE B; STAT PROTEIN; STAT3 PROTEIN;

ANIMAL TISSUE; ARTICLE; C57BL 6 MOUSE; CONTROLLED STUDY; HEART MUSCLE ISCHEMIA; HEART PERFORMANCE; HEART PROTECTION; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; REGULATORY MECHANISM; REPERFUSION INJURY; UPREGULATION; WESTERN BLOTTING;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANIMALS; CHROMONES; CYCLIC S-OXIDES; JANUS KINASES; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MORPHOLINES; MYOCARDIAL CONTRACTION; MYOCARDIAL REPERFUSION INJURY; MYOCYTES, CARDIAC; PHOSPHORYLATION; PROTEIN KINASE INHIBITORS; PROTO-ONCOGENE PROTEINS C-AKT; RECOVERY OF FUNCTION; SIGNAL TRANSDUCTION; STAT3 TRANSCRIPTION FACTOR; TIME FACTORS; TYRPHOSTINS; VENTRICULAR FUNCTION, LEFT;

EID: 57049143783     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00692.2008     Document Type: Article

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