What makes the mitochondria a killer? Can we condition them to be less destructive?

Biochimica et Biophysica Acta - Molecular Cell Research

Volumn 1813, Issue 7, 2011, Pages 1302-1308

  Murphy, Elizabeth ^a   Steenbergen, Charles ^b  

^a NATIONAL HEART LUNG AND BLOOD INSTITUTE   (United States)

^b JHMI   (United States)

Author keywords

Calcium; Cardioprotection; Mitochondria; Nitric oxide; Phosphorylation; Reactive oxygen species

Indexed keywords

CALCIUM; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; NITRIC OXIDE; REACTIVE OXYGEN METABOLITE;

APOPTOSIS; CELL DEATH; HEART PROTECTION; HUMAN; MITOCHONDRIAL MEMBRANE; MITOCHONDRIAL PERMEABILITY; MITOCHONDRION; NONHUMAN; PRIORITY JOURNAL; PROTEIN PROCESSING; REPERFUSION INJURY; REVIEW; SIGNAL TRANSDUCTION;

ANIMALS; CALCIUM; CELL DEATH; ISCHEMIC PRECONDITIONING, MYOCARDIAL; MICE; MITOCHONDRIA, HEART; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MYOCARDIAL REPERFUSION INJURY; MYOCYTES, CARDIAC; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION;

EID: 79957942134     PISSN: 01674889     EISSN: 18792596     Source Type: Journal    
DOI: 10.1016/j.bbamcr.2010.09.003     Document Type: Review

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