APP homodimers transduce an amyloid-β-mediated increase in release probability at excitatory synapses

Cell Reports

Volumn 7, Issue 5, 2014, Pages 1560-1576

  Fogel, Hilla ^a   Frere, Samuel ^a   Segev, Oshik ^a   Bharill, Shashank ^b   Shapira, Ilana ^a   Gazit, Neta ^a   O'Malley, Tiernan ^c,d   Slomowitz, Edden ^a   Berdichevsky, Yevgeny ^a   Walsh, DominicM ^d   Isacoff, EhudY ^b   Hirsch, JoelA ^a   Slutsky, Inna ^a  

^a TEL AVIV UNIVERSITY   (Israel)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c UNIVERSITY COLLEGE DUBLIN   (Ireland)

^d Brigham and Women's Hospital Center for Neurologic Diseases   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN[1-40]; AMYLOID PRECURSOR PROTEIN; CALCIUM; GLUTAMIC ACID; HOMODIMER; INHIBITORY GUANINE NUCLEOTIDE BINDING PROTEIN; MONOMER; PRESYNAPTIC RECEPTOR; AMYLOID BETA PROTEIN;

ADULT; ALZHEIMER DISEASE; ANIMAL CELL; ARTICLE; CALCIUM TRANSPORT; CELL MEMBRANE; CONFORMATIONAL TRANSITION; CONTROLLED STUDY; FEEDBACK SYSTEM; HIPPOCAMPUS; MOUSE; NERVE CELL STIMULATION; NEWBORN; NONHUMAN; PRESYNAPTIC NERVE; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN MODIFICATION; PROTEIN PROTEIN INTERACTION; RAT; REGULATORY MECHANISM; SIGNAL TRANSDUCTION; SYNAPSE VESICLE; SYNAPTIC TRANSMISSION; XENOPUS; ANIMAL TISSUE; BINDING AFFINITY; COMPLEX FORMATION; EXCITATORY SYNAPSE; NEUROPATHOLOGY; NEUROTRANSMITTER RELEASE; POSITIVE FEEDBACK; PROTEIN CONFORMATION; PROTEIN SECRETION; SYNAPSE;

EID: 84902332604     PISSN: None     EISSN: 22111247     Source Type: Journal    
DOI: 10.1016/j.celrep.2014.04.024     Document Type: Article

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