Disruption of Ion Homeostasis in the Neurogliovascular Unit Underlies the Pathogenesis of Ischemic Cerebral Edema

Translational Stroke Research

Volumn 5, Issue 1, 2014, Pages 3-16

  Khanna, Arjun ^a,b   Kahle, Kristopher T ^a,b   Walcott, Brian P ^a,b   Gerzanich, Volodymyr ^c   Simard, J Marc ^c  

^a HARVARD MEDICAL SCHOOL   (United States)

^b MASSACHUSETTS GENERAL HOSPITAL   (United States)

^c UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

Author keywords

Blood brain barrier; Bumetanide; Edema; Glyburide; Hemorrhage; Hypertonic; Ion channel; Ischemia; NKCC1; Stroke; SUR1; TRPM4; Tumor

Indexed keywords

ACID SENSING ION CHANNEL; AQUAPORIN; ION; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; SODIUM POTASSIUM CHLORIDE COTRANSPORTER 1; SODIUM PROTON EXCHANGE PROTEIN; TRANSIENT RECEPTOR POTENTIAL CHANNEL; TRANSIENT RECEPTOR POTENTIAL CHANNEL M4;

APOPTOSIS; ARTICLE; BLOOD VESSEL TONE; BRAIN BLOOD VESSEL; BRAIN EDEMA; BRAIN ISCHEMIA; CELL PROLIFERATION; CELL VOLUME; EXTRACELLULAR SPACE; GAP JUNCTION; HOMEOSTASIS; HUMAN; ION CURRENT; ION TRANSPORT; NEUROGLIOVASCULAR UNIT; NONHUMAN; PATHOGENESIS; PATHOPHYSIOLOGY; PRIORITY JOURNAL;

BRAIN EDEMA; BRAIN ISCHEMIA; CEREBROVASCULAR CIRCULATION; ENDOTHELIAL CELLS; HOMEOSTASIS; HUMANS; ION TRANSPORT; MEMBRANE TRANSPORT PROTEINS; NEUROGLIA;

EID: 84893807007     PISSN: 18684483     EISSN: 1868601X     Source Type: Journal    
DOI: 10.1007/s12975-013-0307-9     Document Type: Article

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