Crosstalk between Bcl-2 family and Ras family small GTPases: potential cell fate regulation?

Frontiers in Oncology

Volumn 2 JAN, Issue , 2013, Pages

  Kang, Jia ^a,b   Pervaiz, Shazib ^a,b,c,d  

^a NATIONAL UNIVERSITY OF SINGAPORE   (Singapore)

^b NATIONAL UNIVERSITY OF SINGAPORE   (Singapore)

^c DUKE NUS MEDICAL SCHOOL   (Singapore)

^d MASSACHUSETTS INSTITUTE OF TECHNOLOGY   (United States)

Author keywords

Apoptosis; BCL 2 family; Interaction; Ras family small GTPases; Redox

Indexed keywords

ARF PROTEIN; CISPLATIN; DOXORUBICIN; ETOPOSIDE; GEFITINIB; GUANOSINE TRIPHOSPHATASE; HYPOXIA INDUCIBLE FACTOR 1; IMATINIB; MEVINOLIN; MITOGEN ACTIVATED PROTEIN KINASE; PACLITAXEL; PROTEIN BCL 2; PROTEIN BCL XL; PROTEIN KINASE C; RAB PROTEIN; RAC PROTEIN; RAC1 PROTEIN; RAF PROTEIN; RAN PROTEIN; RAP PROTEIN; RAS PROTEIN; REACTIVE OXYGEN METABOLITE; RHEB PROTEIN; RHO KINASE; STRESS ACTIVATED PROTEIN KINASE; SULINDAC;

ACUTE LYMPHOBLASTIC LEUKEMIA; APOPTOSIS; BREAST CANCER; CELL DEATH; CELL FATE; CELL PERMEABILIZATION; CELL PROLIFERATION; CELLULAR DISTRIBUTION; DOWN REGULATION; DRUG INACTIVATION; GENE OVEREXPRESSION; HUMAN; LUNG NON SMALL CELL CANCER; OVARY CANCER; PROSTATE CANCER; PROTEIN PROCESSING; PROTEIN SYNTHESIS REGULATION; REVIEW; SIGNAL TRANSDUCTION; UBIQUITINATION;

EID: 84878889924     PISSN: None     EISSN: 2234943X     Source Type: Journal    
DOI: 10.3389/fonc.2012.00206     Document Type: Review

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