Enhanced β-secretase processing alters APP axonal transport and leads to axonal defects

Human Molecular Genetics

Volumn 21, Issue 21, 2012, Pages 4587-4601

  Rodrigues, Elizabeth M ^a   Weissmiller, April M ^b,c   Goldstein, Lawrence S B ^b,c,d  

^a UNIVERSITY OF CALIFORNIA SAN DIEGO   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c UNIVERSITY OF CALIFORNIA   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID PRECURSOR PROTEIN; BETA SECRETASE; BETA SECRETASE INHIBITOR; GAMMA SECRETASE INHIBITOR;

AMYLOID PLAQUE; ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; FAMILIAL DISEASE; HYPOTHESIS; IN VIVO STUDY; MOUSE; MUTATION; NERVE FIBER TRANSPORT; NEUROAXONAL DYSTROPHY; NEWBORN; NONHUMAN; PRIORITY JOURNAL; PROTEIN CLEAVAGE; SWEDEN;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; AMYLOID PRECURSOR PROTEIN SECRETASES; ANIMALS; AXONAL TRANSPORT; CELLS, CULTURED; ENZYME INHIBITORS; GENE EXPRESSION REGULATION; HUMANS; MALE; MICE; MUTATION; NEURONS;

EID: 84867766569     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/dds297     Document Type: Article

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