Axonal accumulation of synaptic markers in APP transgenic Drosophila depends on the NPTY motif and is paralleled by defects in synaptic plasticity

European Journal of Neuroscience

Volumn 25, Issue 4, 2007, Pages 1079-1086

  Rusu, Patricia ^a,b   Jansen, Anna ^b   Soba, Peter ^a,c   Kirsch, Joachim ^a   Löwer, Alexander ^a   Merdes, Gunter ^a   Kuan, Yung Hui ^a   Jung, Anita ^b   Beyreuther, Konrad ^a   Kjaerulff, Ole ^b   Kins, Stefan ^a  

^a UNIVERSITY OF HEIDELBERG   (Germany)

^b UNIVERSITY OF COPENHAGEN   (Denmark)

^c UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

APPL; Axonopathies; Axonopathy; Neurodegenerative disease; Neuromuscular junction

Indexed keywords

AMYLOID PRECURSOR PROTEIN;

ALZHEIMER DISEASE; ARTICLE; COGNITION; DROSOPHILA MELANOGASTER; LARVA; NERVE CELL PLASTICITY; NERVE FIBER; NERVE FIBER TRANSPORT; NEUROMUSCULAR SYNAPSE; NEUROTRANSMISSION; NONHUMAN; PERIPHERAL NERVE; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN EXPRESSION; PROTEIN INTERACTION; SYNAPTIC TRANSMISSION; TRANSGENIC ORGANISM;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; AMINO ACID MOTIFS; AMYLOID BETA-PROTEIN PRECURSOR; ANALYSIS OF VARIANCE; ANIMALS; ANIMALS, GENETICALLY MODIFIED; AXONS; DROSOPHILA MELANOGASTER; GENE EXPRESSION REGULATION; GREEN FLUORESCENT PROTEINS; HUMANS; LARVA; MICE; MUTAGENESIS; NERVE TISSUE PROTEINS; NEUROMUSCULAR JUNCTION; NUCLEAR PROTEINS; SYNAPTOTAGMINS;

EID: 33847194657     PISSN: 0953816X     EISSN: 14609568     Source Type: Journal    
DOI: 10.1111/j.1460-9568.2007.05341.x     Document Type: Article

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