Hypoxia suppresses conversion from proliferative arrest to cellular senescence

Proceedings of the National Academy of Sciences of the United States of America

Volumn 109, Issue 33, 2012, Pages 13314-13318

  Leontieva, Olga V ^a   Natarajan, Venkatesh ^a   Demidenko, Zoya N ^a   Burdelya, Lyudmila G ^a   Gudkov, Andrei V ^a   Blagosklonny, Mikhail V ^a  

^a ROSWELL PARK CANCER INSTITUTE   (United States)

Author keywords

Biology; Gerontology; Oncology

Indexed keywords

ETOPOSIDE; HYPOXIA INDUCIBLE FACTOR 1; HYPOXIA INDUCIBLE FACTOR 1ALPHA; MAMMALIAN TARGET OF RAPAMYCIN; NUTLIN 3; PROTEIN P21; PROTEIN P53;

ARTICLE; CANCER CELL; CELL CYCLE ARREST; CELL PROLIFERATION; CELL REGENERATION; CELL STRUCTURE; CONTROLLED STUDY; DNA DAMAGE; FIBROBLAST; HUMAN; HUMAN CELL; HYPOXIA; PRIORITY JOURNAL; PROTEIN EXPRESSION; RETINA CELL; SENESCENCE;

CELL AGING; CELL HYPOXIA; CELL LINE; CELL PROLIFERATION; CYCLIN-DEPENDENT KINASE INHIBITOR P21; EPITHELIAL CELLS; ETOPOSIDE; FIBROBLASTS; HUMANS; HYPOXIA-INDUCIBLE FACTOR 1, ALPHA SUBUNIT; IMIDAZOLES; PIPERAZINES; TUMOR SUPPRESSOR PROTEIN P53;

EID: 84865173843     PISSN: 00278424     EISSN: 10916490     Source Type: Journal    
DOI: 10.1073/pnas.1205690109     Document Type: Article

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