Elimination of proliferating cells unmasks the shift from senescence to quiescence caused by rapamycin

PLoS ONE

Volumn 6, Issue 10, 2011, Pages

  Leontieva, Olga V ^a   Demidenko, Zoya N ^a   Gudkov, Andrei V ^a   Blagosklonny, Mikhail V ^a  

^a ROSWELL PARK CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MAMMALIAN TARGET OF RAPAMYCIN; NOCODAZOLE; NUTLIN 3; PROTEIN P21; PROTEIN P53; RAPAMYCIN; IMIDAZOLE DERIVATIVE; ISOPROPYL THIOGALACTOSIDE; PIPERAZINE DERIVATIVE;

ARTICLE; CELL AGING; CELL CYCLE ARREST; CELL PROLIFERATION; CELL STRUCTURE; CELLULAR PARAMETERS; CONTROLLED STUDY; HUMAN; HUMAN CELL; PHENOTYPE; PROTEIN FUNCTION; PROTEIN INTERACTION; QUIESCENCE; CELL SHAPE; DRUG EFFECT; TUMOR CELL LINE;

CELL AGING; CELL LINE, TUMOR; CELL PROLIFERATION; CELL SHAPE; HUMANS; IMIDAZOLES; ISOPROPYL THIOGALACTOSIDE; NOCODAZOLE; PIPERAZINES; SIROLIMUS;

EID: 80054005335     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0026126     Document Type: Article

References (17)

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