Nitric oxide and calcium signaling regulate myocardial tumor necrosis factor-α expression and cardiac function in sepsis

Canadian Journal of Physiology and Pharmacology

Volumn 88, Issue 2, 2010, Pages 92-104

  Zhang, Ting ^a   Feng, Qingping ^a  

^a UNIVERSITY OF WESTERN ONTARIO   (Canada)

Author keywords

Calcium; Cardiac dysfunction; Nitric oxide; Sepsis; Signal transduction; Tumor necrosis factor

Indexed keywords

CALCIUM ION; ENDOTHELIAL NITRIC OXIDE SYNTHASE; GLYCOGEN SYNTHASE KINASE 3ALPHA; GLYCOGEN SYNTHASE KINASE 3BETA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; LIPOPOLYSACCHARIDE; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; MITOGEN ACTIVATED PROTEIN KINASE P38; NEURONAL NITRIC OXIDE SYNTHASE; NITRIC OXIDE; PHOSPHOLIPASE C GAMMA1; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE 2; STAT PROTEIN; STRESS ACTIVATED PROTEIN KINASE 1; SUPPRESSOR OF CYTOKINE SIGNALING 1; TUMOR NECROSIS FACTOR ALPHA;

CALCIUM SIGNALING; CONFERENCE PAPER; ENZYME PHOSPHORYLATION; HEART FUNCTION; HEART MUSCLE CELL; NONHUMAN; PRIORITY JOURNAL; SEPSIS;

ANIMALS; CALCIUM SIGNALING; GENE EXPRESSION REGULATION; HEART; HUMANS; MYOCARDIUM; NITRIC OXIDE; SEPSIS; TUMOR NECROSIS FACTOR-ALPHA;

EID: 77649123845     PISSN: 00084212     EISSN: None     Source Type: Journal    
DOI: 10.1139/Y09-097     Document Type: Conference Paper

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