Protection against lipopolysacharide-induced myocardial dysfunction in mice by cardiac-specific expression of soluble Fas

Journal of Molecular and Cellular Cardiology

Volumn 44, Issue 1, 2008, Pages 160-169

  Niu, Jianli ^a   Azfer, Asim ^a   Kolattukudy, Pappachan E ^a  

^a UNIVERSITY OF CENTRAL FLORIDA COLLEGE OF MEDICINE   (United States)

Author keywords

Fas FasL signaling; Lipopolysacharide; Myocardial dysfunction; Sepsis

Indexed keywords

CASPASE 3; CASPASE 7; FAS ANTIGEN; FAS LIGAND; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INDUCIBLE NITRIC OXIDE SYNTHASE; INTERLEUKIN 1BETA; INTERLEUKIN 6; LIPOPOLYSACCHARIDE; PEROXYNITRITE; SODIUM CHLORIDE; TOLL LIKE RECEPTOR 4; TUMOR NECROSIS FACTOR ALPHA; 3 NITROTYROSINE; 3-NITROTYROSINE; CYTOKINE; DRUG DERIVATIVE; FAS PROTEIN, MOUSE; TLR4 PROTEIN, MOUSE; TYROSINE; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CYTOKINE PRODUCTION; ECHOCARDIOGRAPHY; ENZYME ACTIVITY; HEART FUNCTION; HEART LEFT VENTRICLE EJECTION FRACTION; HEART MUSCLE; HEART PROTECTION; INFLAMMATION; LUNG FLUID; LUNG INJURY; MALE; MOUSE; MYOCARDIAL DISEASE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PROTEIN INTERACTION; SEPSIS; SIGNAL TRANSDUCTION; TRANSCRIPTION REGULATION; TRANSGENIC MOUSE; UPREGULATION; WATER CONTENT; WILD TYPE; ANIMAL; ANTIBODY SPECIFICITY; BIOSYNTHESIS; CELL DEATH; CHEMICALLY INDUCED DISORDER; DRUG EFFECT; ENZYME ACTIVATION; GENE EXPRESSION REGULATION; GENETICS; HEART; METABOLISM; PATHOPHYSIOLOGY; SOLUBILITY;

MUS; MUS MUSCULUS;

ANIMALS; ANTIGENS, CD95; CASPASE 3; CASPASE 7; CELL DEATH; CYTOKINES; ECHOCARDIOGRAPHY; ENZYME ACTIVATION; GENE EXPRESSION REGULATION; HEART; LIPOPOLYSACCHARIDES; MALE; MICE; MYOCARDIUM; NF-KAPPA B; NITRIC OXIDE SYNTHASE TYPE II; ORGAN SPECIFICITY; SEPSIS; SOLUBILITY; TOLL-LIKE RECEPTOR 4; TYROSINE;

EID: 37549071105     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2007.09.016     Document Type: Article

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