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Volumn 98, Issue 21, 2001, Pages 12283-12288
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The in vivo role of p38 MAP kinases in cardiac remodeling and restrictive cardiomyopathy
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Author keywords
Conditional transgenesis; Heart failure
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Indexed keywords
MITOGEN ACTIVATED PROTEIN KINASE;
SYNAPTOPHYSIN;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
CONTROLLED STUDY;
DEATH;
DIASTOLE;
ECHOCARDIOGRAPHY;
ENZYME ACTIVATION;
ENZYME ACTIVITY;
ENZYME INDUCTION;
GENE EXPRESSION;
HEART;
HEART CONTRACTION;
HEART FAILURE;
HEART MUSCLE CELL;
HEART MUSCLE FIBROSIS;
HEART VENTRICLE;
HEART VENTRICLE REMODELING;
HEART VENTRICLE SIZE;
INOTROPISM;
MARKER GENE;
NONHUMAN;
PRESSURE VOLUME CURVE;
PRIORITY JOURNAL;
RAT;
RESTRICTIVE CARDIOMYOPATHY;
SYSTOLE;
TRANSGENIC ANIMAL;
ANIMALS;
CA(2+)-CALMODULIN DEPENDENT PROTEIN KINASE;
CARDIOMYOPATHY, RESTRICTIVE;
CARDIOTONIC AGENTS;
CELLS, CULTURED;
GENE EXPRESSION;
GENE TARGETING;
HEART VENTRICLES;
HEMODYNAMIC PROCESSES;
HUMANS;
MAP KINASE KINASE 3;
MAP KINASE KINASE 6;
MICE;
MICE, TRANSGENIC;
MITOGEN-ACTIVATED PROTEIN KINASE KINASES;
MITOGEN-ACTIVATED PROTEIN KINASES;
P38 MITOGEN-ACTIVATED PROTEIN KINASES;
PROTEIN-TYROSINE KINASES;
RATS;
VENTRICULAR REMODELING;
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EID: 0035834071
PISSN: 00278424
EISSN: None
Source Type: Journal
DOI: 10.1073/pnas.211086598 Document Type: Article |
Times cited : (300)
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References (38)
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