Caspase-3 deficiency during development increases vulnerability to hypoxic-ischemic injury through caspase-3-independent pathways

Neurobiology of Disease

Volumn 22, Issue 3, 2006, Pages 523-537

  West, Tim ^a   Atzeva, Madeliene ^a   Holtzman, David M ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; Brain injury; Casapse 3; Cell death; DNA laddering; Hypoxia ischemia; Neonatal

Indexed keywords

CASPASE 3; OXYGEN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN ISCHEMIA; CAROTID ARTERY LIGATION; CASPASE 3 DEFICIENCY; CELL NUCLEUS; CELL STRUCTURE; CONTROLLED STUDY; DISEASE PREDISPOSITION; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ENZYME DEFICIENCY; ENZYME INHIBITION; EXPOSURE; FEMALE; MALE; MOUSE; NERVE CELL NECROSIS; NEWBORN; NEWBORN HYPOXIA; NONHUMAN; PERINATAL DEVELOPMENT; PERINATAL PERIOD; PRIORITY JOURNAL; PROTEIN FUNCTION; REGULATORY MECHANISM; WILD TYPE;

ANIMALS; ANIMALS, NEWBORN; BLOTTING, WESTERN; BRAIN; CASPASE 3; CASPASES; CELL NUCLEUS; HYPOXIA-ISCHEMIA, BRAIN; IN SITU NICK-END LABELING; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; POLYMERASE CHAIN REACTION; RATS;

EID: 33744805129     PISSN: 09699961     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.nbd.2005.12.017     Document Type: Article

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