Increase in the ratio of mitochondrial Bax/Bcl-XL induces Bax activation in human leukemic K562 cell line

Apoptosis

Volumn 9, Issue 3, 2004, Pages 377-384

  Liu, F T ^a   Goff, L K ^a   Hao, J H ^a   Newland, A C ^a   Jia, L ^a  

^a QUEEN MARY UNIVERSITY OF LONDON   (United Kingdom)

Author keywords

Bax activation; Bcl XL translocation; DNA damage; Mitochondria; Ratio of Bax Bcl XL

Indexed keywords

BH3 PROTEIN; CYTOCHROME C; PROTEIN BAX; PROTEIN BCL 2; PROTEIN BCL XL; PROTEIN P53;

APOPTOSIS; ARTICLE; CELL FREE SYSTEM; CELL STRAIN K 562; CONFORMATIONAL TRANSITION; CONTROLLED STUDY; DIMERIZATION; GENE TRANSLOCATION; GENETIC TRANSFECTION; HUMAN; HUMAN CELL; LEUKEMIA CELL; MITOCHONDRION; OLIGOMERIZATION; PRIORITY JOURNAL; ULTRAVIOLET RADIATION;

APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; BCL-X PROTEIN; BH3 INTERACTING DOMAIN DEATH AGONIST PROTEIN; CARRIER PROTEINS; CYTOCHROME C GROUP; HUMANS; K562 CELLS; KINETICS; MITOCHONDRIA; PROTEIN CONFORMATION; PROTEIN TRANSPORT; PROTO-ONCOGENE PROTEINS C-BCL-2; TRANSFECTION; ULTRAVIOLET RAYS;

HOMO;

EID: 3242763808     PISSN: 13608185     EISSN: None     Source Type: Journal    
DOI: 10.1023/B:APPT.0000025815.78761.5c     Document Type: Article

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