Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion

Biochemical Journal

Volumn 390, Issue 2, 2005, Pages 377-394

  Solaini, Giancarlo ^a,c   Harris, David A ^b  

^a SCUOLA SUPERIORE SANT ANNA   (Italy)

^b UNIVERSITY OF OXFORD   (United Kingdom)

^c UNIVERSITY OF BOLOGNA   (Italy)

Author keywords

F1F0 ATPase; Ischaemia; Mitochondria; Reactive oxygen species (ROS); Reperfusion

Indexed keywords

ADENOSINETRIPHOSPHATE; ANTIOXIDANTS; BIODIVERSITY; CARDIOLOGY; ENZYMES; OXYGEN; POSITIVE IONS; TISSUE;

F1F0-ATPASE; ISCHAEMIA; MITOCHONDRIA; REACTIVE OXYGEN SPECIES (ROS); REPERFUSION;

BIOCHEMISTRY;

2,4 DINITROPHENOL; 5 HYDROXYDECANOIC ACID; AMILORIDE; AMOBARBITAL; ANESTHETIC AGENT; CARNITINE DERIVATIVE; CASPASE 3 INHIBITOR; CHLORAMPHENICOL; CYCLOSPORIN; DIAZOXIDE; DICHLOROACETIC ACID; DILTIAZEM; DIPYRIDAMOLE; GALLOPAMIL; ION CHANNEL; MALONIC ACID; MYXOTHIAZOL; NICORANDIL; NICOTINIC ACID; NISOLDIPINE; OLIGOMYCIN; OXFENICINE; PROPOFOL; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE; RANOLAZINE; ROTENONE; RUTHENIUM RED; SEVOFLURANE; TETRODOTOXIN; TRIMETAZIDINE; UNINDEXED DRUG;

APOPTOSIS; ELECTRON TRANSPORT; ENZYME ACTIVITY; HEART FUNCTION; HEART MITOCHONDRION; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; HEART MUSCLE METABOLISM; HEART MUSCLE REPERFUSION; HEART PROTECTION; MEMBRANE PERMEABILITY; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; MITOCHONDRIAL RESPIRATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN SYNTHESIS INHIBITION; REVIEW;

ANIMALS; CARDIOVASCULAR AGENTS; HUMANS; ION CHANNELS; MITOCHONDRIA, HEART; MYOCARDIAL REPERFUSION INJURY;

EID: 25144478337     PISSN: 02646021     EISSN: None     Source Type: Journal    
DOI: 10.1042/BJ20042006     Document Type: Review

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