Dinitrophenol, cyclosporin A, and trimetazidine modulate preconditioning in the isolated rat heart: Support for a mitochondrial role in cardioprotection

Cardiovascular Research

Volumn 47, Issue 1, 2000, Pages 68-73

  Minners, Jan ^a   Van Den Bos, Ewout J ^a   Yellon, Derek M ^b   Schwalb, Herzl ^c   Opie, Lionel H ^a   Sack, Michael N ^a  

^a Hatter Institute for Cardiovascular Research   (South Africa)

^b UNIVERSITY COLLEGE LONDON HOSPITALS   (United Kingdom)

^c HADASSAH HEBREW UNIVERSITY MEDICAL CENTER   (Israel)

Author keywords

Infarction; Ischemia; K ATP channel; Mitochondria; Oxidative phosphorylation; Preconditioning

Indexed keywords

2,4 DINITROPHENOL; CYCLOSPORIN A; TRIMETAZIDINE;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DRUG EFFECT; DRUG SCREENING; HEART INFARCTION SIZE; HEART MUSCLE ISCHEMIA; HEART PROTECTION; HEMODYNAMICS; ISOLATED HEART; MALE; NONHUMAN; PRIORITY JOURNAL; RAT;

ANIMALS; ANTI-ARRHYTHMIA AGENTS; CYCLOSPORINE; DECANOIC ACIDS; DINITROPHENOLS; ENZYME INHIBITORS; HOMEOSTASIS; HYDROXY ACIDS; ISCHEMIC PRECONDITIONING, MYOCARDIAL; MALE; MITOCHONDRIA, HEART; MYOCARDIAL ISCHEMIA; PERFUSION; RATS; RATS, LONG-EVANS; TRIMETAZIDINE; UNCOUPLING AGENTS;

EID: 0034044807     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0008-6363(00)00069-9     Document Type: Article

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