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Proceedings of the National Academy of Sciences of the United States of America

Volumn 99, Issue 13, 2002, Pages 8892-8897

Inhibition of mitochondrial respiration by endogenous nitric oxide: A critical step in Fas signaling

(6) Beltrán, Belén a Quintero, Marisol a García Zaragozá, Eugenia b O'Connor, Enrique b Esplugues, Juan V b Moncada, Salvador c

a CENTRO NACIONAL DE INVESTIGACIONES CARDIOVASCULARES CNIC (Spain)

b UNIVERSITY OF VALENCIA (Spain)

c UNIVERSITY COLLEGE LONDON (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATE; CASPASE 8; FAS ANTIGEN; FREE RADICAL; NITRIC OXIDE; NITRIC OXIDE SYNTHASE INHIBITOR; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE; SUPEROXIDE;

ARTICLE; CELL DEATH; CELL SURVIVAL; CONTROLLED STUDY; ENZYME ACTIVATION; GLYCOLYSIS; HUMAN; HUMAN CELL; HYDROLYSIS; HYPERPOLARIZATION; LEUKEMIA CELL LINE; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; MITOCHONDRIAL RESPIRATION; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANTIGENS, CD95; APOPTOSIS; CASPASE 8; CASPASE 9; CASPASES; CELL SURVIVAL; HUMANS; JURKAT CELLS; MEMBRANE POTENTIALS; MITOCHONDRIA; NITRIC OXIDE; OXYGEN CONSUMPTION; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION;

EID: 0037172971 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.092259799 Document Type: Article

Times cited : (118)

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