Focus on Kir6.2: A key component of the ATP-sensitive potassium channel

Journal of Molecular and Cellular Cardiology

Volumn 38, Issue 6, 2005, Pages 927-936

  Haider, Shozeb ^a   Antcliff, Jennifer F ^a   Proks, Peter ^a   Sansom, Mark S P ^a   Ashcroft, Frances M ^a  

^a UNIVERSITY OF OXFORD   (United Kingdom)

Author keywords

ATP sensitive K+ channel; Kir6.2, insulin secretion

Indexed keywords

ADENOSINE TRIPHOSPHATE; CALCIUM; GLIBENCLAMIDE; GLICLAZIDE; GLUCOSE; INSULIN; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR6.2; POTASSIUM CHANNEL; POTASSIUM ION; SULFONYLUREA; SULFONYLUREA RECEPTOR; VOLTAGE GATED CALCIUM CHANNEL;

AMINO TERMINAL SEQUENCE; AUTOSOMAL RECESSIVE INHERITANCE; BINDING SITE; CARBOXY TERMINAL SEQUENCE; CHANNEL GATING; DIABETES MELLITUS; ELECTRIC ACTIVITY; ELECTROCARDIOGRAM; GENE MUTATION; HUMAN; HYPERINSULINISM; IC 50; INSULIN RELEASE; LIGAND BINDING; NEUROLOGIC DISEASE; NON INSULIN DEPENDENT DIABETES MELLITUS; PANCREAS ISLET BETA CELL; PERSISTENT HYPERINSULINEMIC HYPOGLYCEMIA OF INFANCY; PRIORITY JOURNAL; REVIEW; SYMPTOM;

ADENOSINE TRIPHOSPHATE; ANIMALS; BINDING SITES; BLOOD GLUCOSE; DIABETES MELLITUS, TYPE 2; DIMERIZATION; GLUCOSE; HUMANS; INFANT, NEWBORN; INSULIN; MODELS, MOLECULAR; MUTATION; NERVOUS SYSTEM DISEASES; POLYMORPHISM, GENETIC; POTASSIUM; POTASSIUM CHANNELS; POTASSIUM CHANNELS, INWARDLY RECTIFYING; PROTEIN CONFORMATION; PROTEIN STRUCTURE, TERTIARY; STRUCTURE-ACTIVITY RELATIONSHIP;

EID: 19444367729     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2005.01.007     Document Type: Review

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