Insights into extracellular matrix functions from mutant mouse models

Experimental Cell Research

Volumn 261, Issue 1, 2000, Pages 52-68

  Gustafsson, Erika ^a   Fässler, Reinhard ^a  

^a LUND UNIVERSITY   (Sweden)

Author keywords

Basement membranes; Embryogenesis; Extracellular matrix; Gene targeting; Mouse

Indexed keywords

DISCOIDIN; DYSTROGLYCAN; INTEGRIN; TYROSINE KINASE RECEPTOR;

ANIMAL MODEL; BASEMENT MEMBRANE; BRAIN DEVELOPMENT; EMBRYO DEVELOPMENT; EXTRACELLULAR MATRIX; GENE MUTATION; GENE TARGETING; HEART DEVELOPMENT; HUMAN; KIDNEY DEVELOPMENT; MOUSE; NONHUMAN; OSSIFICATION; PHENOTYPE; PRIORITY JOURNAL; SHORT SURVEY; THROMBOCYTE FUNCTION; WOUND HEALING;

ANIMALIA;

EID: 0034715896     PISSN: 00144827     EISSN: None     Source Type: Journal    
DOI: 10.1006/excr.2000.5042     Document Type: Article

References (162)

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103. β4 Integrin is required for hemidesmosome formation, cell adhesion and cell survival
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126. Multiple negative elements in a gene that codes for an extracellular matrix protein, collagen X, restrict expression to hypertrophic chondrocytes
127. Mutations in the gene encoding the human matrix Gla protein cause Keutel syndrome
128. Targeted disruption of the biglycan gene leads to an osteoporosis-like phenotype in mice
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131. Increased marrow-derived osteoprogenitor cells and endosteal bone formation in mice lacking thrombospondin 2
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