Mice that lack the angiogenesis inhibitor, thrombospondin 2, mount an altered foreign body reaction characterized by increased vascularity

Proceedings of the National Academy of Sciences of the United States of America

Volumn 96, Issue 8, 1999, Pages 4449-4454

  Kyriakides, Themis R ^a   Leach, Kathleen J ^b   Hoffman, Allan S ^b   Ratner, Buddy D ^b,c   Bornstein, Paul ^a  

^a UNIVERSITY OF WASHINGTON SCHOOL OF MEDICINE   (United States)

^b University of Washington   (United States)

^c University of Washington   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANGIOGENESIS INHIBITOR; THROMBOSPONDIN; THROMBOSPONDIN 2; UNCLASSIFIED DRUG;

ANIMAL TISSUE; ARTICLE; BLOOD VESSEL; CONNECTIVE TISSUE; EXTRACELLULAR MATRIX; FIBROBLAST; FOREIGN BODY REACTION; GENE DISRUPTION; IMMUNE RESPONSE; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; VASCULARIZATION;

ANIMALS; CELL ADHESION MOLECULES; CELLS, CULTURED; DIMETHYLPOLYSILOXANES; EXTRACELLULAR MATRIX; FIBROBLASTS; FOREIGN-BODY REACTION; MICE; MICE, KNOCKOUT; NEOVASCULARIZATION, PATHOLOGIC; OXIDATION-REDUCTION; SILICONES; SKIN; THROMBOSPONDINS;

ANIMALIA;

EID: 0033551072     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.96.8.4449     Document Type: Article

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