Prion-like transmission of pathogenic protein aggregates in genetic models of neurodegenerative disease

Current Opinion in Genetics and Development

Volumn 44, Issue , 2017, Pages 149-155

  Pearce, Margaret MP ^a  

^a UNIVERSITY OF THE SCIENCES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA SYNUCLEIN; AMYLOID PRECURSOR PROTEIN; COPPER ZINC SUPEROXIDE DISMUTASE; HUNTINGTIN; POLYGLUTAMINE; TAU PROTEIN;

ALZHEIMER DISEASE; AMYLOID PLAQUE; AMYOTROPHIC LATERAL SCLEROSIS; ARTIFICIAL NEURAL NETWORK; BRAIN REGION; CELL LOSS; CELL POPULATION; DEGENERATIVE DISEASE; GENE MUTATION; GENETIC MODEL; GENOME-WIDE ASSOCIATION STUDY; GLIA CELL; HUMAN; NERVE CELL; NONHUMAN; PATHOGENESIS; PHAGOCYTOSIS; PROTEIN AGGREGATION; PROTEIN CLEAVAGE; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; PROTEIN MISFOLDING; PROTEIN SECRETION; PROTEIN TRANSPORT; REVIEW; RISK FACTOR; SYNAPSE; TAUOPATHY; GENETICS; METABOLISM; PATHOLOGY; PRION; PRION DISEASE; PROTEINOSIS; TRANSMISSION;

HUMANS; NEURODEGENERATIVE DISEASES; NEURONS; PRION DISEASES; PRIONS; PROTEIN AGGREGATION, PATHOLOGICAL;

EID: 85018509589     PISSN: 0959437X     EISSN: 18790380     Source Type: Journal    
DOI: 10.1016/j.gde.2017.03.011     Document Type: Review

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