Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells

Journal of Cell Biology

Volumn 208, Issue 5, 2015, Pages 563-579

  Zellweger, Ralph ^a   Dalcher, Damian ^a   Mutreja, Karun ^a   Berti, Matteo ^b   Schmid, Jonas A ^a   Herrador, Raquel ^a   Vindigni, Alessandro ^b   Lopes, Massimo ^a  

^a UNIVERSITY OF ZURICH   (Switzerland)

^b SAINT LOUIS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DNA TOPOISOMERASE; POLY(ADENOSINE DIPHOSPHATE RIBOSE); RAD51 PROTEIN; SINGLE STRANDED DNA; DNA TOPOISOMERASE INHIBITOR; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; RAD51 PROTEIN, HUMAN; RECQ HELICASE; RECQL PROTEIN, HUMAN;

ARTICLE; CANCER CHEMOTHERAPY; CELL STRESS; CHROMOSOME BREAKAGE; CROSS LINKING; DNA REPLICATION; DNA STRAND BREAKAGE; DNA SYNTHESIS; DRUG TARGETING; ELECTRON MICROSCOPY; GENOTOXICITY; HOMOLOGOUS RECOMBINATION; HUMAN; HUMAN CELL; NONHUMAN; PRIORITY JOURNAL; REPLISOME; DNA DAMAGE; GENETICS; HEK293 CELL LINE; METABOLISM; TUMOR CELL LINE;

CELL LINE, TUMOR; DNA DAMAGE; DNA REPLICATION; HEK293 CELLS; HUMANS; POLY(ADP-RIBOSE) POLYMERASES; RAD51 RECOMBINASE; RECQ HELICASES; TOPOISOMERASE INHIBITORS;

EID: 84924911767     PISSN: 00219525     EISSN: 15408140     Source Type: Journal    
DOI: 10.1083/jcb.201406099     Document Type: Article

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