UPR, autophagy, and mitochondria crosstalk underlies the ER stress response

Trends in Biochemical Sciences

Volumn 40, Issue 3, 2015, Pages 141-148

  Senft, Daniela ^a   Ronai, Ze'ev A ^a  

^a BURNHAM INSTITUTE   (United States)

Author keywords

Autophagy; ER stress; Hypoxia; Mitochondria; Ubiquitin; UPR

Indexed keywords

ACTIVATING TRANSCRIPTION FACTOR 4; ACTIVATING TRANSCRIPTION FACTOR 6; BECLIN 1; GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153; INITIATION FACTOR 2ALPHA; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN IRE1; PROTEIN KINASE R; STRESS ACTIVATED PROTEIN KINASE; SUMO PROTEIN; TUMOR NECROSIS FACTOR RECEPTOR ASSOCIATED FACTOR 2; UBIQUITIN PROTEIN LIGASE; X BOX BINDING PROTEIN 1;

AUTOPHAGY; CELL DEATH; CELL HYPOXIA; CELL STRESS; CELL SURVIVAL; ENDOPLASMIC RETICULUM ASSOCIATED DEGRADATION; ENDOPLASMIC RETICULUM STRESS; ENZYME ACTIVITY; HUMAN; INFLAMMATION; MITOCHONDRION; MOLECULAR INTERACTION; MOUSE; NONHUMAN; OXIDATIVE STRESS; REVIEW; UNFOLDED PROTEIN RESPONSE; ANIMAL; CELL FUNCTION; METABOLISM; PHYSIOLOGICAL STRESS;

ANIMALS; AUTOPHAGY; CELL PHYSIOLOGICAL PHENOMENA; ENDOPLASMIC RETICULUM STRESS; HUMANS; MITOCHONDRIA; STRESS, PHYSIOLOGICAL; UNFOLDED PROTEIN RESPONSE;

EID: 84923195554     PISSN: 09680004     EISSN: 13624326     Source Type: Journal    
DOI: 10.1016/j.tibs.2015.01.002     Document Type: Review

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