Cytoplasmic dynein heavy chain: The servant of many masters

Trends in Neurosciences

Volumn 36, Issue 11, 2013, Pages 641-651

  Schiavo, Giampietro ^a,b   Greensmith, Linda ^a   Hafezparast, Majid ^c   Fisher, Elizabeth M C ^a  

^a NATIONAL HOSPITAL FOR NEUROLOGY AND NEUROSURGERY   (United Kingdom)

^b IMPERIAL CANCER RESEARCH FUND   (United Kingdom)

^c UNIVERSITY OF SUSSEX   (United Kingdom)

Author keywords

Amyotrophic lateral sclerosis; Axonal transport; Cramping 1; Legs at odd angles; Motor neurons; Neurodegeneration; Sprawling

Indexed keywords

BINDING PROTEIN; COPPER ZINC SUPEROXIDE DISMUTASE; CYTOPLASMIC DYNEIN; CYTOPLASMIC DYNEIN HEAVY CHAIN 1; DYNACTIN; MUTANT PROTEIN; PROTEIN BICD1; PROTEIN BICD2; PROTEIN HAP1; PROTEIN HTT; PROTEIN LIS1; PROTEIN NUDE; SNAPIN; UNCLASSIFIED DRUG;

AMYOTROPHIC LATERAL SCLEROSIS; CELL ORGANELLE; CENTRAL NERVOUS SYSTEM; DEGENERATIVE DISEASE; DEVELOPMENTAL DISORDER; GENE DELETION; GENE INACTIVATION; GENE INTERACTION; GENE MUTATION; HEAVY CHAIN; HETEROZYGOTE; HUMAN; LOCOMOTION; MISSENSE MUTATION; MOTOR DYSFUNCTION; NERVE FIBER TRANSPORT; NERVOUS SYSTEM; NERVOUS SYSTEM DEVELOPMENT; NONHUMAN; PERRY SYNDROME; PHENOTYPE; POINT MUTATION; PRIORITY JOURNAL; PROTEIN FUNCTION; REVIEW; SENSORY DYSFUNCTION;

AMYOTROPHIC LATERAL SCLEROSIS; AXONAL TRANSPORT; CRAMPING 1; LEGS AT ODD ANGLES; MOTOR NEURONS; NEURODEGENERATION; SPRAWLING;

ANIMALS; BRAIN; CYTOPLASMIC DYNEINS; GENETIC PREDISPOSITION TO DISEASE; HUMANS; MUTATION; NEURONS; PROTEIN MULTIMERIZATION;

EID: 84886953670     PISSN: 01662236     EISSN: 1878108X     Source Type: Journal    
DOI: 10.1016/j.tins.2013.08.001     Document Type: Review

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