Partial inactivation of cardiac 14-3-3 protein in vivo elicits endoplasmic reticulum stress (ERS) and activates ers-initiated apoptosis in ERS-induced mice

Cellular Physiology and Biochemistry

Volumn 26, Issue 2, 2010, Pages 167-178

  Sari, Flori R ^a,b   Watanabe, Kenichi ^a   Widyantoro, Bambang ^c   Thandavarayan, Rajarajan A ^a   Harima, Meilei ^a   Zhang, Shaosong ^d   Muslin, Anthony J ^e   Kodama, Makoto ^f   Aizawa, Yoshifusa ^f  

^a NIIGATA UNIVERSITY OF PHARMACY AND APPLIED LIFE SCIENCES   (Japan)

^b State Islamic University   (Indonesia)

^c KOBE UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

^d LightLab Imaging Inc   (United States)

^e WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^f NIIGATA UNIVERSITY GRADUATE SCHOOL OF MEDICAL AND DENTAL SCIENCES   (Japan)

Author keywords

14 3 3; Apoptosis; Endoplasmic Reticulum stress; Pressure overload

Indexed keywords

CARRIER PROTEIN; CASPASE 12; CCAAT ENHANCER BINDING PROTEIN; GLUCOSE REGULATED PROTEIN 78; INOSITOL REQUIRING ENZYME 1ALPHA; PROTEIN 14 3 3; PROTEIN 14 3 3 ETA; THAPSIGARGIN; TUMOR NECROSIS FACTOR RECEPTOR ASSOCIATED FACTOR 2; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; ENDOPLASMIC RETICULUM STRESS; HEART LEFT VENTRICLE OVERLOAD; IN VIVO STUDY; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; TRANSGENIC MOUSE;

MUS; MUS MUSCULUS;

EID: 78049343064     PISSN: 10158987     EISSN: None     Source Type: Journal    
DOI: 10.1159/000320548     Document Type: Article

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