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Volumn 106, Issue 2, 2008, Pages 978-988

Depletion of 14-3-3 zeta elicits endoplasmic reticulum stress and cell death, and increases vulnerability to kainate-induced injury in mouse hippocampal cultures

Author keywords

Apoptosis; Epilepsy; Epileptogenesis; Glucose regulated protein 78; Neurodegeneration; Tyrosine 3 mono oxygenase tryptophan 5 monooxygenase activation protein; Unfolded protein response

Indexed keywords

BRAIN PROTEIN; CALNEXIN; KAINIC ACID; PROTEIN 14 3 3 ZETA; SMALL INTERFERING RNA; TUNICAMYCIN; UNCLASSIFIED DRUG; AMINO ACID RECEPTOR STIMULATING AGENT; LYSYL ASPARTYL GLUTAMYL LEUCINE; LYSYL-ASPARTYL-GLUTAMYL-LEUCINE; OLIGOPEPTIDE; PROTEIN 14 3 3; SIGNAL PEPTIDE; VERATRIDINE;

EID: 51649100334     PISSN: 00223042     EISSN: 14714159     Source Type: Journal    
DOI: 10.1111/j.1471-4159.2008.05447.x     Document Type: Article
Times cited : (36)

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