Prolonged endoplasmic reticulum stress in hypertrophic and failing heart after aortic constriction: Possible contribution of endoplasmic reticulum stress to cardiac myocyte apoptosis

Circulation

Volumn 110, Issue 6, 2004, Pages 705-712

  Okada, Ken Ichiro ^a   Minamino, Tetsuo ^a   Tsukamoto, Yoshitane ^b   Liao, Yulin ^a   Tsukamoto, Osamu ^a   Takashima, Seiji ^a   Hirata, Akio ^a   Fujita, Masashi ^a   Nagamachi, Yoko ^a   Nakatani, Takeshi ^d   Yutani, Chikao ^d   Ozawa, Kentaro ^c   Ogawa, Satoshi ^c   Tomoike, Hitonobu ^d   Hori, Masatsugu ^a   Kitakaze, Masafumi ^d  

^a OSAKA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

^b Osaka Medical Center for Cancer   (Japan)

^c KANAZAWA UNIVERSITY   (Japan)

^d NATIONAL CEREBRAL AND CARDIOVASCULAR CENTER   (Japan)

Author keywords

Apoptosis; Endoplasmic reticulum; Heart failure; Hypertrophy

Indexed keywords

ANGIOTENSIN 1 RECEPTOR; BRAIN NATRIURETIC PEPTIDE; CASPASE 12; CASPASE 3; DEOXYURIDINE TRIPHOSPHATE PYROPHOSPHATASE; OLMESARTAN; STRESS ACTIVATED PROTEIN KINASE;

ANIMAL CELL; AORTA CONSTRICTION; APOPTOSIS; ARTICLE; CELL COUNT; CELL CULTURE; CONTROLLED STUDY; DISEASE ACTIVITY; DISEASE COURSE; ECHOCARDIOGRAPHY; ENDOPLASMIC RETICULUM; ENZYME ACTIVATION; ENZYME DEGRADATION; HEART FAILURE; HEART HYPERTROPHY; HEART MUSCLE CELL; HEART STRESS; MALE; MOUSE; NEWBORN; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; UPREGULATION;

ANGIOTENSIN II; ANGIOTENSIN II TYPE 1 RECEPTOR BLOCKERS; ANIMALS; AORTIC VALVE STENOSIS; APOPTOSIS; CARDIOMEGALY; CELLS, CULTURED; DISEASE MODELS, ANIMAL; DISEASE PROGRESSION; ENDOPLASMIC RETICULUM; GENE EXPRESSION REGULATION; HEART FAILURE, CONGESTIVE; HUMANS; IMIDAZOLES; LIGATION; MALE; MICE; MICE, INBRED C57BL; MOLECULAR CHAPERONES; MYOCYTES, CARDIAC; NATRIURETIC PEPTIDE, BRAIN; RATS; RATS, INBRED WKY; RNA, MESSENGER; SIGNAL TRANSDUCTION; STRESS; TETRAZOLES; THAPSIGARGIN; TUNICAMYCIN;

EID: 4043076224     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.0000137836.95625.D4     Document Type: Article

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