Role of cyclophilin D-dependent mitochondrial permeability transition in glutamate-induced calcium deregulation and excitotoxic neuronal death

Experimental Neurology

Volumn 218, Issue 2, 2009, Pages 171-182

  Li, Viacheslav ^a   Brustovetsky, Tatiana ^a   Brustovetsky, Nickolay ^a  

^a INDIANA UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM; CYCLOPHILIN D; GLUTAMIC ACID; YELLOW FLUORESCENT PROTEIN;

ACIDIFICATION; ANIMAL CELL; ARTICLE; BRAIN MITOCHONDRION; BRAIN NERVE CELL; CALCIUM TRANSPORT; CELL DAMAGE; CELL SURVIVAL; CONCENTRATION RESPONSE; CONTROLLED STUDY; DEPOLARIZATION; EXCITOTOXICITY; GENE DELETION; KNOCKOUT MOUSE; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRIAL PERMEABILITY; MOUSE; NERVE CELL NECROSIS; NONHUMAN; PRIORITY JOURNAL; SURVIVAL RATE; WILD TYPE;

ANIMALS; BLOTTING, WESTERN; CALCIUM; CALCIUM SIGNALING; CELL CULTURE TECHNIQUES; CELL DEATH; CELL MEMBRANE PERMEABILITY; CEREBRAL CORTEX; CYCLOPHILINS; DOSE-RESPONSE RELATIONSHIP, DRUG; GENOTYPE; GLUTAMIC ACID; HYDROGEN-ION CONCENTRATION; IMMUNOHISTOCHEMISTRY; LUMINESCENT PROTEINS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICROSCOPY, CONFOCAL; MITOCHONDRIA; MITOCHONDRIAL MEMBRANES; NEURONS; POLYMERASE CHAIN REACTION;

EID: 67649785237     PISSN: 00144886     EISSN: 10902430     Source Type: Journal    
DOI: 10.1016/j.expneurol.2009.02.007     Document Type: Article

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