-
1
-
-
0014140089
-
Patterns of septic shock in man: A detailed study of 56 patients
-
MacLean LD, Mulligan WG, McLean AP, Duff JH. (1967) Patterns of septic shock in man: a detailed study of 56 patients. Ann. Surg. 166:543-62.
-
(1967)
Ann. Surg
, vol.166
, pp. 543-562
-
-
MacLean, L.D.1
Mulligan, W.G.2
McLean, A.P.3
Duff, J.H.4
-
2
-
-
0006328262
-
Bacteremia due to gram-negative bacilli other than the Salmonella; a clinical and therapeutic study
-
Waisbren BA. (1951) Bacteremia due to gram-negative bacilli other than the Salmonella; a clinical and therapeutic study. AMA. Arch. Intern. Med. 88:467-88.
-
(1951)
AMA. Arch. Intern. Med
, vol.88
, pp. 467-488
-
-
Waisbren, B.A.1
-
3
-
-
33646896099
-
Septic shock: A heart story since the 1960s
-
Rabuel C, Mebazaa A. (2006) Septic shock: a heart story since the 1960s. Intensive. Care. Med. 32:799-807.
-
(2006)
Intensive. Care. Med
, vol.32
, pp. 799-807
-
-
Rabuel, C.1
Mebazaa, A.2
-
4
-
-
0013919436
-
Circulatory and metabolic alterations associated with survival or death in peritonitis: Clinical analysis of 25 cases
-
Clowes GH Jr, Vucinic M, Weidner MG. (1966) Circulatory and metabolic alterations associated with survival or death in peritonitis: clinical analysis of 25 cases. Ann. Surg. 163:866-85.
-
(1966)
Ann. Surg
, vol.163
, pp. 866-885
-
-
Clowes Jr, G.H.1
Vucinic, M.2
Weidner, M.G.3
-
5
-
-
0001633299
-
Studies on the circulatory changes in the dog produced by endotoxin from gram-negative microorganisms
-
Maclean LD, Spink WW, Visscher MB, Weil MH. (1956) Studies on the circulatory changes in the dog produced by endotoxin from gram-negative microorganisms. J. Clin. Invest. 35:1191-8.
-
(1956)
J. Clin. Invest
, vol.35
, pp. 1191-1198
-
-
Maclean, L.D.1
Spink, W.W.2
Visscher, M.B.3
Weil, M.H.4
-
6
-
-
0017715012
-
Cardiac depression in bacteremia
-
Postel J, Schloerb PR. (1977) Cardiac depression in bacteremia. Ann. Surg. 186:74-82.
-
(1977)
Ann. Surg
, vol.186
, pp. 74-82
-
-
Postel, J.1
Schloerb, P.R.2
-
7
-
-
0015167278
-
Factors affecting hemodynamics in clinical shock with sepsis
-
Wilson RF, Sarver EJ, LeBlanc PL. (1971) Factors affecting hemodynamics in clinical shock with sepsis. Ann. Surg. 174:939-43.
-
(1971)
Ann. Surg
, vol.174
, pp. 939-943
-
-
Wilson, R.F.1
Sarver, E.J.2
LeBlanc, P.L.3
-
8
-
-
0020960312
-
Optimum left heart filling pressure during fluid resuscitation of patients with hypovolemic and septic shock
-
Packman MI, Rackow EC. (1983) Optimum left heart filling pressure during fluid resuscitation of patients with hypovolemic and septic shock. Crit. Care Med. 11:165-9.
-
(1983)
Crit. Care Med
, vol.11
, pp. 165-169
-
-
Packman, M.I.1
Rackow, E.C.2
-
9
-
-
0015795488
-
Hemodynamic measurements in bacteremia and septic shock in man
-
Gunnar RM, Loeb HS, Winslow EJ, Blain C, Robinson J. (1973) Hemodynamic measurements in bacteremia and septic shock in man. J. Infect. Dis. 128 (Suppl):295-8.
-
(1973)
J. Infect. Dis
, vol.128
, Issue.SUPPL.
, pp. 295-298
-
-
Gunnar, R.M.1
Loeb, H.S.2
Winslow, E.J.3
Blain, C.4
Robinson, J.5
-
10
-
-
0015608557
-
Hemodynamic studies and results of therapy in 50 patients with bacteremic shock
-
Winslow EJ, Loeb HS, Rahimtoola SH, Kamath S, Gunnar RM. (1973) Hemodynamic studies and results of therapy in 50 patients with bacteremic shock. Am. J. Med. 54:421-32.
-
(1973)
Am. J. Med
, vol.54
, pp. 421-432
-
-
Winslow, E.J.1
Loeb, H.S.2
Rahimtoola, S.H.3
Kamath, S.4
Gunnar, R.M.5
-
11
-
-
0017614396
-
Cardiopulmonary effects of volume loading in patients in septic shock
-
Krausz MM, Perel A, Eimerl D, Cotev S. (1977) Cardiopulmonary effects of volume loading in patients in septic shock. Ann. Surg. 185:429-34.
-
(1977)
Ann. Surg
, vol.185
, pp. 429-434
-
-
Krausz, M.M.1
Perel, A.2
Eimerl, D.3
Cotev, S.4
-
12
-
-
0023607880
-
Serial cardiovascular variables in survivors and nonsurvivors of human septic shock: Heart rate as an early predictor of prognosis
-
Parker MM, Shelhamer JH, Natanson C, Alling DW, Parrillo JE. (1987) Serial cardiovascular variables in survivors and nonsurvivors of human septic shock: heart rate as an early predictor of prognosis. Crit. Care Med. 15:923-9.
-
(1987)
Crit. Care Med
, vol.15
, pp. 923-929
-
-
Parker, M.M.1
Shelhamer, J.H.2
Natanson, C.3
Alling, D.W.4
Parrillo, J.E.5
-
13
-
-
0021279377
-
Profound but reversible myocardial depression in patients with septic shock
-
Parker MM, et al. (1984) Profound but reversible myocardial depression in patients with septic shock. Ann. Intern. Med. 100:483-90.
-
(1984)
Ann. Intern. Med
, vol.100
, pp. 483-490
-
-
Parker, M.M.1
-
14
-
-
0025060845
-
Right ventricular dysfunction and dilatation, similar to left ventricular changes, characterize the cardiac depression of septic shock in humans
-
Parker MM, McCarthy KE, Ognibene FP, Parrillo JE. (1990) Right ventricular dysfunction and dilatation, similar to left ventricular changes, characterize the cardiac depression of septic shock in humans. Chest 97:126-31.
-
(1990)
Chest
, vol.97
, pp. 126-131
-
-
Parker, M.M.1
McCarthy, K.E.2
Ognibene, F.P.3
Parrillo, J.E.4
-
15
-
-
0022208356
-
Left ventricular performance in septic shock: Reversible segmental and global abnormalities
-
Ellrodt AG, et al. (1985) Left ventricular performance in septic shock: reversible segmental and global abnormalities. Am. Heart. J. 110:402-9.
-
(1985)
Am. Heart. J
, vol.110
, pp. 402-409
-
-
Ellrodt, A.G.1
-
16
-
-
0024575208
-
Role of endotoxemia in cardiovascular dysfunction and mortality: Escherichia coli and Staphylococcus aureus challenges in a canine model of human septic shock
-
Natanson C, et al. (1989) Role of endotoxemia in cardiovascular dysfunction and mortality: Escherichia coli and Staphylococcus aureus challenges in a canine model of human septic shock. J. Clin. Invest. 83:243-51.
-
(1989)
J. Clin. Invest
, vol.83
, pp. 243-251
-
-
Natanson, C.1
-
17
-
-
0024520830
-
Endotoxin and tumor necrosis factor challenges in dogs simulate the cardiovascular profile of human septic shock
-
Natanson C, et al. (1989) Endotoxin and tumor necrosis factor challenges in dogs simulate the cardiovascular profile of human septic shock. J. Exp. Med. 169:823-32.
-
(1989)
J. Exp. Med
, vol.169
, pp. 823-832
-
-
Natanson, C.1
-
18
-
-
33646773996
-
Diagnostic accuracy of left ventricular function for identifying sepsis among emergency department patients with nontraumatic symptomatic undifferentiated hypotension
-
Jones AE, Craddock PA, Tayal VS, Kline JA. (2005) Diagnostic accuracy of left ventricular function for identifying sepsis among emergency department patients with nontraumatic symptomatic undifferentiated hypotension. Shock 24:513-7.
-
(2005)
Shock
, vol.24
, pp. 513-517
-
-
Jones, A.E.1
Craddock, P.A.2
Tayal, V.S.3
Kline, J.A.4
-
19
-
-
0032725134
-
Persistent preload defect in severe sepsis despite fluid loading: A longitudinal echocardiographic study in patients with septic shock
-
Jardin F, et al. (1999) Persistent preload defect in severe sepsis despite fluid loading: a longitudinal echocardiographic study in patients with septic shock. Chest 116:1354-9.
-
(1999)
Chest
, vol.116
, pp. 1354-1359
-
-
Jardin, F.1
-
20
-
-
0030950888
-
Left ventricular systolic and diastolic function in septic shock
-
Poelaert J, Declerck C, Vogelaers D, Colardyn F, Visser CA. (1997) Left ventricular systolic and diastolic function in septic shock. Intensive Care Med. 23:553-60.
-
(1997)
Intensive Care Med
, vol.23
, pp. 553-560
-
-
Poelaert, J.1
Declerck, C.2
Vogelaers, D.3
Colardyn, F.4
Visser, C.A.5
-
21
-
-
1542614334
-
Brain natriuretic peptide: A marker of myocardial dysfunction and prognosis during severe sepsis
-
Charpentier J, et al. (2004) Brain natriuretic peptide: a marker of myocardial dysfunction and prognosis during severe sepsis. Crit. Care Med. 32:660-5.
-
(2004)
Crit. Care Med
, vol.32
, pp. 660-665
-
-
Charpentier, J.1
-
22
-
-
0022480082
-
The coronary circulation in human septic shock
-
Cunnion RE, Schaer GL, Parker MM, Natanson C, Parrillo JE. (1986) The coronary circulation in human septic shock. Circulation 73:637-44.
-
(1986)
Circulation
, vol.73
, pp. 637-644
-
-
Cunnion, R.E.1
Schaer, G.L.2
Parker, M.M.3
Natanson, C.4
Parrillo, J.E.5
-
23
-
-
0023097510
-
Coronary hemodynamics and myocardial metabolism of lactate, free fatty acids, glucose, and ketones in patients with septic shock
-
Dhainaut JF, et al. (1987) Coronary hemodynamics and myocardial metabolism of lactate, free fatty acids, glucose, and ketones in patients with septic shock. Circulation 75:533-41.
-
(1987)
Circulation
, vol.75
, pp. 533-541
-
-
Dhainaut, J.F.1
-
24
-
-
0034922436
-
Increased troponin in patients with sepsis and septic shock: Myocardial necrosis or reversible myocardial depression?
-
Wu AH. (2001) Increased troponin in patients with sepsis and septic shock: myocardial necrosis or reversible myocardial depression? Intensive Care Med. 27:959-61.
-
(2001)
Intensive Care Med
, vol.27
, pp. 959-961
-
-
Wu, A.H.1
-
25
-
-
0034524327
-
Muscular contractile failure in septic patients: Role of the inducible nitric oxide synthase pathway
-
Lanone S, et al. (2000) Muscular contractile failure in septic patients: role of the inducible nitric oxide synthase pathway. Am. J. Respir. Crit. Care Med. 162:2308-15.
-
(2000)
Am. J. Respir. Crit. Care Med
, vol.162
, pp. 2308-2315
-
-
Lanone, S.1
-
26
-
-
85047680831
-
Maldistribution of heterogeneous coronary blood flow during canine endotoxin shock
-
Groeneveld AB, et al. (1991) Maldistribution of heterogeneous coronary blood flow during canine endotoxin shock. Cardiovasc. Res. 25:80-8.
-
(1991)
Cardiovasc. Res
, vol.25
, pp. 80-88
-
-
Groeneveld, A.B.1
-
27
-
-
0028118993
-
Myocardial energy metabolism and morphology in a canine model of sepsis
-
Solomon MA, et al. (1994) Myocardial energy metabolism and morphology in a canine model of sepsis. Am. J. Physiol. 266:H757-68.
-
(1994)
Am. J. Physiol
, vol.266
-
-
Solomon, M.A.1
-
28
-
-
1842863228
-
Cardiac myocytes activated by septic plasma promote neutrophil transendothelial migration: Role of platelet-activating factor and the chemokines LIX and KC
-
Madorin WS, et al. (2004) Cardiac myocytes activated by septic plasma promote neutrophil transendothelial migration: role of platelet-activating factor and the chemokines LIX and KC. Circ. Res. 94:944-51.
-
(2004)
Circ. Res
, vol.94
, pp. 944-951
-
-
Madorin, W.S.1
-
29
-
-
29744460916
-
Endotoxin-induced heart dysfunction in rats: Assessment of myocardial perfusion and permeability and the role of fluid resuscitation
-
Chagnon F, Bentourkia M, Lecomte R, Lessard M, Lesur O. (2006) Endotoxin-induced heart dysfunction in rats: assessment of myocardial perfusion and permeability and the role of fluid resuscitation. Crit. Care Med. 34:127-33.
-
(2006)
Crit. Care Med
, vol.34
, pp. 127-133
-
-
Chagnon, F.1
Bentourkia, M.2
Lecomte, R.3
Lessard, M.4
Lesur, O.5
-
30
-
-
0030982851
-
Myocardial collagen changes and edema in rats with hyperdynamic sepsis
-
Yu P, et al. (1997) Myocardial collagen changes and edema in rats with hyperdynamic sepsis. Crit. Care Med. 25:657-62.
-
(1997)
Crit. Care Med
, vol.25
, pp. 657-662
-
-
Yu, P.1
-
32
-
-
0015028384
-
Relationship between splanchnic blood flow and a myocardial depressant factor in endotoxin shock
-
Wangensteen SL, Geissinger WT, Lovett WL, Glenn TM, Lefer AM. (1971) Relationship between splanchnic blood flow and a myocardial depressant factor in endotoxin shock. Surgery 69:410-8.
-
(1971)
Surgery
, vol.69
, pp. 410-418
-
-
Wangensteen, S.L.1
Geissinger, W.T.2
Lovett, W.L.3
Glenn, T.M.4
Lefer, A.M.5
-
33
-
-
0022409016
-
A circulating myocardial depressant substance in humans with septic shock: Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance
-
Parrillo JE, et al. (1985) A circulating myocardial depressant substance in humans with septic shock: septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance. J. Clin. Invest. 76:1539-53.
-
(1985)
J. Clin. Invest
, vol.76
, pp. 1539-1553
-
-
Parrillo, J.E.1
-
34
-
-
0029975877
-
Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum
-
Kumar A, et al. (1996) Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum. J. Exp. Med. 183:949-58.
-
(1996)
J. Exp. Med
, vol.183
, pp. 949-958
-
-
Kumar, A.1
-
35
-
-
0026735208
-
Negative inotropic effects of cytokines on the heart mediated by nitric oxide
-
Finkel MS, et al. (1992) Negative inotropic effects of cytokines on the heart mediated by nitric oxide. Science 257:387-9.
-
(1992)
Science
, vol.257
, pp. 387-389
-
-
Finkel, M.S.1
-
36
-
-
31344464342
-
An essential role for complement C5a in the pathogenesis of septic cardiac dysfunction
-
Niederbichler AD, et al. (2006) An essential role for complement C5a in the pathogenesis of septic cardiac dysfunction. J. Exp. Med. 203:53-61.
-
(2006)
J. Exp. Med
, vol.203
, pp. 53-61
-
-
Niederbichler, A.D.1
-
37
-
-
18344384146
-
Tumor necrosis factor-alpha-induced caspase activation mediates endotoxin-related cardiac dysfunction
-
Carlson DL, Willis MS, White DJ, Horton JW, Giroir BP. (2005) Tumor necrosis factor-alpha-induced caspase activation mediates endotoxin-related cardiac dysfunction. Crit. Care Med. 33:1021-8.
-
(2005)
Crit. Care Med
, vol.33
, pp. 1021-1028
-
-
Carlson, D.L.1
Willis, M.S.2
White, D.J.3
Horton, J.W.4
Giroir, B.P.5
-
38
-
-
0344009602
-
Macrophage migration inhibitory factor is a cardiac-derived myocardial depressant factor
-
Garner LB, et al. (2003) Macrophage migration inhibitory factor is a cardiac-derived myocardial depressant factor. Am. J. Physiol. Heart. Circ. Physiol. 285:H2500-9.
-
(2003)
Am. J. Physiol. Heart. Circ. Physiol
, vol.285
-
-
Garner, L.B.1
-
39
-
-
0036781708
-
IL-1beta and IL-6 act synergistically with TNF-alpha to alter cardiac contractile function after burn trauma
-
Maass DL, White J, Horton JW. (2002) IL-1beta and IL-6 act synergistically with TNF-alpha to alter cardiac contractile function after burn trauma. Shock 18:360-6.
-
(2002)
Shock
, vol.18
, pp. 360-366
-
-
Maass, D.L.1
White, J.2
Horton, J.W.3
-
40
-
-
33645744996
-
Regulation of lung inflammation in the model of IgG immune-complex injury
-
Gao H, Neff T, Ward P.A. (2006) Regulation of lung inflammation in the model of IgG immune-complex injury. Annu. Rev. Pathol. Mech. Dis. 1:215-42.
-
(2006)
Annu. Rev. Pathol. Mech. Dis
, vol.1
, pp. 215-242
-
-
Gao, H.1
Neff, T.2
Ward, P.A.3
-
41
-
-
0037105426
-
Complement-induced impairment of innate immunity during sepsis
-
Huber-Lang MS, et al. (2002) Complement-induced impairment of innate immunity during sepsis. J. Immunol. 169:3223-31.
-
(2002)
J. Immunol
, vol.169
, pp. 3223-3231
-
-
Huber-Lang, M.S.1
-
42
-
-
0035863922
-
Role of C5a in multiorgan failure during sepsis
-
Huber-Lang M, et al. (2001) Role of C5a in multiorgan failure during sepsis. J. Immunol. 166:1193-9.
-
(2001)
J. Immunol
, vol.166
, pp. 1193-1199
-
-
Huber-Lang, M.1
-
43
-
-
0036615932
-
C5a receptor and thymocyte apoptosis in sepsis
-
Riedemann NC, et al. (2002) C5a receptor and thymocyte apoptosis in sepsis. FASEB J. 16:887-8.
-
(2002)
FASEB J
, vol.16
, pp. 887-888
-
-
Riedemann, N.C.1
-
44
-
-
0036092494
-
Anti-c5a ameliorates coagulation/ fibrinolytic protein changes in a rat model of sepsis
-
Laudes IJ, et al. (2002) Anti-c5a ameliorates coagulation/ fibrinolytic protein changes in a rat model of sepsis. Am. J. Pathol. 160:1867-75.
-
(2002)
Am. J. Pathol
, vol.160
, pp. 1867-1875
-
-
Laudes, I.J.1
-
45
-
-
0032884049
-
Sequential changes in autonomic regulation of cardiac myocytes after in vivo endotoxin injection in rat
-
Abi-Gerges N, et al. (1999) Sequential changes in autonomic regulation of cardiac myocytes after in vivo endotoxin injection in rat. Am. J. Respir. Crit. Care. Med. 160:1196-204.
-
(1999)
Am. J. Respir. Crit. Care. Med
, vol.160
, pp. 1196-1204
-
-
Abi-Gerges, N.1
-
46
-
-
0035909920
-
Activation of cardiac endothelium as a compensatory component in endotoxin-induced cardiomyopathy: Role of endothelin, prostaglandins, and nitric oxide
-
Mebazaa A, et al. (2001) Activation of cardiac endothelium as a compensatory component in endotoxin-induced cardiomyopathy: role of endothelin, prostaglandins, and nitric oxide. Circulation 104:3137-44.
-
(2001)
Circulation
, vol.104
, pp. 3137-3144
-
-
Mebazaa, A.1
-
47
-
-
0027255030
-
Oxygen consumption and resting metabolic rate in sepsis, sepsis syndrome, and septic shock
-
Kreymann G, et al. (1993) Oxygen consumption and resting metabolic rate in sepsis, sepsis syndrome, and septic shock. Crit. Care Med. 21:1012-9.
-
(1993)
Crit. Care Med
, vol.21
, pp. 1012-1019
-
-
Kreymann, G.1
-
48
-
-
34249326963
-
Mechanisms of sepsis-induced cardiac dysfunction
-
Rudiger A, Singer M. (2007) Mechanisms of sepsis-induced cardiac dysfunction. Crit. Care Med. 35:1599-608.
-
(2007)
Crit. Care Med
, vol.35
, pp. 1599-1608
-
-
Rudiger, A.1
Singer, M.2
-
50
-
-
0026012502
-
Peripheral oxygen availability within skeletal muscle in sepsis and septic shock: Comparison to limited infection and cardiogenic shock
-
Boekstegers P, Weidenhofer S, Pilz G, Werdan K. (1991) Peripheral oxygen availability within skeletal muscle in sepsis and septic shock: comparison to limited infection and cardiogenic shock. Infection 19:317-23.
-
(1991)
Infection
, vol.19
, pp. 317-323
-
-
Boekstegers, P.1
Weidenhofer, S.2
Pilz, G.3
Werdan, K.4
-
51
-
-
33845690127
-
Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction
-
Rossi MA, Celes MR, Prado CM, Saggioro FP. (2007) Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction. Shock 27:10-8.
-
(2007)
Shock
, vol.27
, pp. 10-18
-
-
Rossi, M.A.1
Celes, M.R.2
Prado, C.M.3
Saggioro, F.P.4
-
52
-
-
29144530137
-
Evidence of myocardial hibernation in the septic heart
-
Levy RJ, et al. (2005) Evidence of myocardial hibernation in the septic heart. Crit. Care Med. 33:2752-6.
-
(2005)
Crit. Care Med
, vol.33
, pp. 2752-2756
-
-
Levy, R.J.1
-
53
-
-
10744222951
-
Apoptosis of neurons in cardiovascular autonomic centres triggered by inducible nitric oxide synthase after death from septic shock
-
Sharshar T, et al. (2003) Apoptosis of neurons in cardiovascular autonomic centres triggered by inducible nitric oxide synthase after death from septic shock. Lancet 362:1799-805.
-
(2003)
Lancet
, vol.362
, pp. 1799-1805
-
-
Sharshar, T.1
-
54
-
-
1042265202
-
The neuropathology of septic shock
-
Sharshar T, et al. (2004) The neuropathology of septic shock. Brain Pathol. 14:21-33.
-
(2004)
Brain Pathol
, vol.14
, pp. 21-33
-
-
Sharshar, T.1
-
55
-
-
0032778367
-
Inappropriate sympathetic activation at onset of septic shock: A spectral analysis approach
-
Annane D, et al. (1999) Inappropriate sympathetic activation at onset of septic shock: a spectral analysis approach. Am. J. Respir. Crit. Care Med. 160:458-65.
-
(1999)
Am. J. Respir. Crit. Care Med
, vol.160
, pp. 458-465
-
-
Annane, D.1
-
56
-
-
0034948109
-
Cardiac variability in critically ill adults: Influence of sepsis
-
Korach M, et al. (2001) Cardiac variability in critically ill adults: influence of sepsis. Crit. Care Med. 29:1380-5.
-
(2001)
Crit. Care Med
, vol.29
, pp. 1380-1385
-
-
Korach, M.1
-
58
-
-
0034713266
-
Vagus nerve stimulation attenuates the systemic inflammatory response to endotoxin
-
Borovikova LV, et al. (2000) Vagus nerve stimulation attenuates the systemic inflammatory response to endotoxin. Nature 405:458-62.
-
(2000)
Nature
, vol.405
, pp. 458-462
-
-
Borovikova, L.V.1
-
59
-
-
36448948021
-
Transcutaneous vagus nerve stimulation reduces serum high mobility group box 1 levels and improves survival in murine sepsis
-
Huston JM, et al. (2007) Transcutaneous vagus nerve stimulation reduces serum high mobility group box 1 levels and improves survival in murine sepsis. Crit. Care Med. 35: 2762-8.
-
(2007)
Crit. Care Med
, vol.35
, pp. 2762-2768
-
-
Huston, J.M.1
-
60
-
-
0037180778
-
The inflammatory reflex
-
Tracey KJ. (2002) The inflammatory reflex. Nature 420:853-9.
-
(2002)
Nature
, vol.420
, pp. 853-859
-
-
Tracey, K.J.1
-
61
-
-
36549008718
-
Reduced L-type calcium current in ventricular myocytes from endotoxemic guinea pigs
-
Zhong J, Hwang TC, Adams HR, Rubin LJ. (1997) Reduced L-type calcium current in ventricular myocytes from endotoxemic guinea pigs. Am. J. Physiol. 273:H2312-24.
-
(1997)
Am. J. Physiol
, vol.273
-
-
Zhong, J.1
Hwang, T.C.2
Adams, H.R.3
Rubin, L.J.4
-
62
-
-
0028940807
-
2+ current by interleukin-1 beta in cultured rat ventricular myocytes
-
2+ current by interleukin-1 beta in cultured rat ventricular myocytes. Am. J. Physiol. 268:C339-49.
-
(1995)
Am. J. Physiol
, vol.268
-
-
Liu, S.1
Schreur, K.D.2
-
63
-
-
0030002538
-
Endotoxin-induced cardiac depression is associated with decreased cardiac dihydropyridine receptors in rabbits
-
Lew WY, Yasuda S, Yuan T, Hammond HK. (1996) Endotoxin-induced cardiac depression is associated with decreased cardiac dihydropyridine receptors in rabbits. J. Mol. Cell Cardiol. 28:1367-71.
-
(1996)
J. Mol. Cell Cardiol
, vol.28
, pp. 1367-1371
-
-
Lew, W.Y.1
Yasuda, S.2
Yuan, T.3
Hammond, H.K.4
-
64
-
-
0032076172
-
Myofilament calcium sensitivity is decreased in skinned cardiac fibres of endotoxin-treated rabbits
-
Tavernier B, Garrigue D, Boulle C, Vallet B, Adnet P. (1998) Myofilament calcium sensitivity is decreased in skinned cardiac fibres of endotoxin-treated rabbits. Cardiovasc. Res. 38:472-9.
-
(1998)
Cardiovasc. Res
, vol.38
, pp. 472-479
-
-
Tavernier, B.1
Garrigue, D.2
Boulle, C.3
Vallet, B.4
Adnet, P.5
-
65
-
-
0035138303
-
2+ sensitivity but normal mitochondrial function in septic heart
-
2+ sensitivity but normal mitochondrial function in septic heart. Am. J. Respir. Crit. Care Med. 163:362-7.
-
(2001)
Am. J. Respir. Crit. Care Med
, vol.163
, pp. 362-367
-
-
Tavernier, B.1
-
66
-
-
0029940216
-
Paracrine modulation of heart cell function by endothelial cells
-
Shah AM. (1996) Paracrine modulation of heart cell function by endothelial cells. Cardiovasc. Res. 31:847-67.
-
(1996)
Cardiovasc. Res
, vol.31
, pp. 847-867
-
-
Shah, A.M.1
-
67
-
-
0035403179
-
Impairment of the ryanodine-sensitive calcium release channels in the cardiac sarcoplasmic reticulum and its underlying mechanism during the hypodynamic phase of sepsis
-
Dong LW, Wu LL, Ji Y, Liu MS. (2001) Impairment of the ryanodine-sensitive calcium release channels in the cardiac sarcoplasmic reticulum and its underlying mechanism during the hypodynamic phase of sepsis. Shock 16:33-9.
-
(2001)
Shock
, vol.16
, pp. 33-39
-
-
Dong, L.W.1
Wu, L.L.2
Ji, Y.3
Liu, M.S.4
-
68
-
-
0036167330
-
Dual site phospholamban phosphorylation and its physiological relevance in the heart
-
Hagemann D, Xiao RP. (2002) Dual site phospholamban phosphorylation and its physiological relevance in the heart. Trends Cardiovasc. Med. 12:51-6.
-
(2002)
Trends Cardiovasc. Med
, vol.12
, pp. 51-56
-
-
Hagemann, D.1
Xiao, R.P.2
-
70
-
-
0035234205
-
Calcium uptake by sarcoplasmic reticulum is impaired during the hypodynamic phase of sepsis in the rat heart
-
Wu LL, Ji Y, Dong LW, Liu MS. (2001) Calcium uptake by sarcoplasmic reticulum is impaired during the hypodynamic phase of sepsis in the rat heart. Shock 15:49-55.
-
(2001)
Shock
, vol.15
, pp. 49-55
-
-
Wu, L.L.1
Ji, Y.2
Dong, L.W.3
Liu, M.S.4
-
71
-
-
0036579768
-
Altered phospholamban-calcium ATPase interaction in cardiac sarcoplasmic reticulum during the progression of sepsis
-
Wu LL, Tang C, Dong LW, Liu MS. (2002) Altered phospholamban-calcium ATPase interaction in cardiac sarcoplasmic reticulum during the progression of sepsis. Shock 17:389-93.
-
(2002)
Shock
, vol.17
, pp. 389-393
-
-
Wu, L.L.1
Tang, C.2
Dong, L.W.3
Liu, M.S.4
-
73
-
-
0032701642
-
Toll4 (TLR4) expression in cardiac myocytes in normal and failing myocardium
-
Frantz S, et al. (1999) Toll4 (TLR4) expression in cardiac myocytes in normal and failing myocardium. J. Clin. Invest. 104:271-80.
-
(1999)
J. Clin. Invest
, vol.104
, pp. 271-280
-
-
Frantz, S.1
-
75
-
-
0035895913
-
Role of TLR-2 in the activation of nuclear factor kappaB by oxidative stress in cardiac myocytes
-
Frantz S, Kelly RA, Bourcier T. (2001) Role of TLR-2 in the activation of nuclear factor kappaB by oxidative stress in cardiac myocytes. J. Biol. Chem. 276:5197-203.
-
(2001)
J. Biol. Chem
, vol.276
, pp. 5197-5203
-
-
Frantz, S.1
Kelly, R.A.2
Bourcier, T.3
-
76
-
-
0036086439
-
Escherichia coli LPS-induced LV dysfunction: Role of Toll-like receptor-4 in the adult heart
-
Nemoto S, et al. (2002) Escherichia coli LPS-induced LV dysfunction: role of Toll-like receptor-4 in the adult heart. Am. J. Physiol. Heart Circ. Physiol. 282:H2316-23.
-
(2002)
Am. J. Physiol. Heart Circ. Physiol
, vol.282
-
-
Nemoto, S.1
-
77
-
-
0037623904
-
IRAK1 deletion disrupts cardiac Toll/IL-1 signaling and protects against contractile dysfunction
-
Thomas JA, et al. (2003) IRAK1 deletion disrupts cardiac Toll/IL-1 signaling and protects against contractile dysfunction. Am. J. Physiol. Heart Circ. Physiol. 285:H597-606.
-
(2003)
Am. J. Physiol. Heart Circ. Physiol
, vol.285
-
-
Thomas, J.A.1
-
78
-
-
0035371187
-
In vivo expression of proinflammatory mediators in the adult heart after endotoxin administration: The role of Toll-like receptor-4
-
Baumgarten G, et al. (2001) In vivo expression of proinflammatory mediators in the adult heart after endotoxin administration: the role of Toll-like receptor-4. J. Infect. Dis. 183:1617-24.
-
(2001)
J. Infect. Dis
, vol.183
, pp. 1617-1624
-
-
Baumgarten, G.1
-
79
-
-
0027407363
-
Transgenic mice expressing human CD14 are hypersensitive to lipopolysaccharide
-
Ferrero E, et al. (1993) Transgenic mice expressing human CD14 are hypersensitive to lipopolysaccharide. Proc. Natl. Acad. Sci. U. S. A. 90:2380-4.
-
(1993)
Proc. Natl. Acad. Sci. U. S. A
, vol.90
, pp. 2380-2384
-
-
Ferrero, E.1
-
80
-
-
0029919991
-
Resistance to endotoxin shock and reduced dissemination of gram-negative bacteria in CD14-deficient mice
-
Haziot A, et al. (1996) Resistance to endotoxin shock and reduced dissemination of gram-negative bacteria in CD14-deficient mice. Immunity 4:407-14.
-
(1996)
Immunity
, vol.4
, pp. 407-414
-
-
Haziot, A.1
-
81
-
-
0037069420
-
CD14-deficient mice are protected against lipopolysaccharide-induced cardiac inflammation and left ventricular dysfunction
-
Knuefermann P, et al. (2002) CD14-deficient mice are protected against lipopolysaccharide-induced cardiac inflammation and left ventricular dysfunction. Circulation 106:2608-15.
-
(2002)
Circulation
, vol.106
, pp. 2608-2615
-
-
Knuefermann, P.1
-
83
-
-
0018775975
-
Plasma catecholamine levels and pulmonary dysfunction in sepsis
-
Bocking JK, Sibbald WJ, Holliday RL, Scott S, Viidik T. (1979) Plasma catecholamine levels and pulmonary dysfunction in sepsis. Surg. Gynecol. Obstet. 148:715-9.
-
(1979)
Surg. Gynecol. Obstet
, vol.148
, pp. 715-719
-
-
Bocking, J.K.1
Sibbald, W.J.2
Holliday, R.L.3
Scott, S.4
Viidik, T.5
-
84
-
-
0031674214
-
Beta-adrenergic receptor-dependent and -independent stimulation of adenylate cyclase is impaired during severe sepsis in humans
-
Bernardin G, Strosberg AD, Bernard A, Mattei M, Marullo S. (1998) Beta-adrenergic receptor-dependent and -independent stimulation of adenylate cyclase is impaired during severe sepsis in humans. Intensive Care Med. 24:1315-22.
-
(1998)
Intensive Care Med
, vol.24
, pp. 1315-1322
-
-
Bernardin, G.1
Strosberg, A.D.2
Bernard, A.3
Mattei, M.4
Marullo, S.5
-
85
-
-
0029382098
-
Sustained elevation in circulating catecholamine levels during polymicrobial sepsis
-
Hahn PY, et al. (1995) Sustained elevation in circulating catecholamine levels during polymicrobial sepsis. Shock 4:269-73.
-
(1995)
Shock
, vol.4
, pp. 269-273
-
-
Hahn, P.Y.1
-
86
-
-
0035103292
-
Cardiac functional and structural alterations induced by endotoxin in rats: Importance of platelet-activating factor
-
Iwase M, et al. (2001) Cardiac functional and structural alterations induced by endotoxin in rats: importance of platelet-activating factor. Crit. Care Med. 29:609-17.
-
(2001)
Crit. Care Med
, vol.29
, pp. 609-617
-
-
Iwase, M.1
-
87
-
-
0030049454
-
Initial externalization followed by internalization of beta-adrenergic receptors in rat heart during sepsis
-
Tang C, Liu MS. (1996) Initial externalization followed by internalization of beta-adrenergic receptors in rat heart during sepsis. Am. J. Physiol. 270:R254-63.
-
(1996)
Am. J. Physiol
, vol.270
-
-
Tang, C.1
Liu, M.S.2
-
88
-
-
0023122079
-
Myocardial adrenergic responsiveness after lethal and nonlethal doses of endotoxin
-
Shepherd RE, Lang CH, McDonough KH. (1987) Myocardial adrenergic responsiveness after lethal and nonlethal doses of endotoxin. Am. J. Physiol. 252:H410-6.
-
(1987)
Am. J. Physiol
, vol.252
-
-
Shepherd, R.E.1
Lang, C.H.2
McDonough, K.H.3
-
89
-
-
0343875590
-
Interleukin 1 and tumor necrosis factor inhibit cardiac myocyte beta-adrenergic responsiveness
-
Gulick T, Chung MK, Pieper SJ, Lange LG, Schreiner GF. (1989) Interleukin 1 and tumor necrosis factor inhibit cardiac myocyte beta-adrenergic responsiveness. Proc. Natl. Acad. Sci. U. S. A. 86:6753-7.
-
(1989)
Proc. Natl. Acad. Sci. U. S. A
, vol.86
, pp. 6753-6757
-
-
Gulick, T.1
Chung, M.K.2
Pieper, S.J.3
Lange, L.G.4
Schreiner, G.F.5
-
90
-
-
0033636934
-
Impairment of cardiac beta-adrenoceptor cellular signaling by decreased expression of G(s alpha) in septic rabbits
-
Matsuda N, et al. (2000) Impairment of cardiac beta-adrenoceptor cellular signaling by decreased expression of G(s alpha) in septic rabbits. Anesthesiology. 93:1465-73.
-
(2000)
Anesthesiology
, vol.93
, pp. 1465-1473
-
-
Matsuda, N.1
-
91
-
-
1542647136
-
G protein and adenylate cyclase complex-mediated signal transduction in the rat heart during sepsis
-
Wu LL, et al. (2003) G protein and adenylate cyclase complex-mediated signal transduction in the rat heart during sepsis. Shock 19:533-7.
-
(2003)
Shock
, vol.19
, pp. 533-537
-
-
Wu, L.L.1
-
92
-
-
0028855687
-
Increase of myocardial inhibitory G-proteins in catecholamine-refractory septic shock or in septic multiorgan failure
-
Bohm M, Kirchmayr R, Gierschik P, Erdmann E. (1995) Increase of myocardial inhibitory G-proteins in catecholamine-refractory septic shock or in septic multiorgan failure. Am. J. Med. 98:183-6.
-
(1995)
Am. J. Med
, vol.98
, pp. 183-186
-
-
Bohm, M.1
Kirchmayr, R.2
Gierschik, P.3
Erdmann, E.4
-
93
-
-
0035066383
-
Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation
-
Kyriakis JM, Avruch J. (2001) Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation. Physiol. Rev. 81:807-69.
-
(2001)
Physiol. Rev
, vol.81
, pp. 807-869
-
-
Kyriakis, J.M.1
Avruch, J.2
-
94
-
-
11144307292
-
STRESS signaling pathways that modulate cardiac myocyte apoptosis
-
Baines CP, Molkentin JD. (2005) STRESS signaling pathways that modulate cardiac myocyte apoptosis. J. Mol. Cell. Cardiol. 38:47-62.
-
(2005)
J. Mol. Cell. Cardiol
, vol.38
, pp. 47-62
-
-
Baines, C.P.1
Molkentin, J.D.2
-
95
-
-
2542433090
-
Protein phosphatase 2A-mediated cross-talk between p38 MAPK and ERK in apoptosis of cardiac myocytes
-
Liu Q, Hofmann PA. (2004) Protein phosphatase 2A-mediated cross-talk between p38 MAPK and ERK in apoptosis of cardiac myocytes. Am. J. Physiol. Heart. Circ. Physiol. 286:H2204-12.
-
(2004)
Am. J. Physiol. Heart. Circ. Physiol
, vol.286
-
-
Liu, Q.1
Hofmann, P.A.2
-
96
-
-
0030036727
-
Stimulation of the stress-activated mitogen-activated protein kinase subfamilies in perfused heart: P38/RK mitogen-activated protein kinases and c-Jun N-terminal kinases are activated by ischemia/reperfusion
-
Bogoyevitch MA, et al. (1996) Stimulation of the stress-activated mitogen-activated protein kinase subfamilies in perfused heart: p38/RK mitogen-activated protein kinases and c-Jun N-terminal kinases are activated by ischemia/reperfusion. Circ. Res. 79:162-73.
-
(1996)
Circ. Res
, vol.79
, pp. 162-173
-
-
Bogoyevitch, M.A.1
-
97
-
-
0033179153
-
Activation of c-Jun N-terminal kinases and p38-mitogen-activated protein kinases in human heart failure secondary to ischaemic heart disease
-
Cook SA, Sugden PH, Clerk A. (1999) Activation of c-Jun N-terminal kinases and p38-mitogen-activated protein kinases in human heart failure secondary to ischaemic heart disease. J. Mol. Cell Cardiol. 31:1429-34.
-
(1999)
J. Mol. Cell Cardiol
, vol.31
, pp. 1429-1434
-
-
Cook, S.A.1
Sugden, P.H.2
Clerk, A.3
-
98
-
-
0035800880
-
Matrix metalloproteinase inhibition after myocardial infarction: A new approach to prevent heart failure?
-
Creemers EE, Cleutjens JP, Smits JF, Daemen MJ. (2001) Matrix metalloproteinase inhibition after myocardial infarction: a new approach to prevent heart failure? Circ. Res. 89:201-10.
-
(2001)
Circ. Res
, vol.89
, pp. 201-210
-
-
Creemers, E.E.1
Cleutjens, J.P.2
Smits, J.F.3
Daemen, M.J.4
-
99
-
-
0024243962
-
Inadequate collagen tethers in dilated cardiopathy
-
Weber KT, Pick R, Janicki JS, Gadodia G, Lakier JB. (1988) Inadequate collagen tethers in dilated cardiopathy. Am. Heart. J. 116:1641-6.
-
(1988)
Am. Heart. J
, vol.116
, pp. 1641-1646
-
-
Weber, K.T.1
Pick, R.2
Janicki, J.S.3
Gadodia, G.4
Lakier, J.B.5
-
100
-
-
0029978342
-
Remodeling of human myocardial collagen in idiopathic dilated cardiomyopathy: Role of metalloproteinases and pyridinoline cross-links
-
Gunja-Smith Z, Morales AR, Romanelli R, Woessner JF Jr. (1996) Remodeling of human myocardial collagen in idiopathic dilated cardiomyopathy: role of metalloproteinases and pyridinoline cross-links. Am. J. Pathol. 148:1639-48.
-
(1996)
Am. J. Pathol
, vol.148
, pp. 1639-1648
-
-
Gunja-Smith, Z.1
Morales, A.R.2
Romanelli, R.3
Woessner Jr., J.F.4
-
101
-
-
0037155057
-
Matrix metalloproteinases in vascular remodeling and atherogenesis: The good, the bad, and the ugly
-
Galis ZS, Khatri JJ. (2002) Matrix metalloproteinases in vascular remodeling and atherogenesis: the good, the bad, and the ugly. Circ. Res. 90:251-62.
-
(2002)
Circ. Res
, vol.90
, pp. 251-262
-
-
Galis, Z.S.1
Khatri, J.J.2
-
102
-
-
0034681937
-
Matrix metalloproteinase-2 contributes to ischemia-reperfusion injury in the heart
-
Cheung PY, et al. (2000) Matrix metalloproteinase-2 contributes to ischemia-reperfusion injury in the heart. Circulation 101:1833-9.
-
(2000)
Circulation
, vol.101
, pp. 1833-1839
-
-
Cheung, P.Y.1
-
103
-
-
0037126045
-
Intracellular action of matrix metalloproteinase-2 accounts for acute myocardial ischemia and reperfusion injury
-
Wang W, et al. (2002) Intracellular action of matrix metalloproteinase-2 accounts for acute myocardial ischemia and reperfusion injury. Circulation 106:1543-9.
-
(2002)
Circulation
, vol.106
, pp. 1543-1549
-
-
Wang, W.1
-
104
-
-
0141839852
-
Matrix metalloproteinase inhibitors attenuate endotoxemia induced cardiac dysfunction: A potential role for MMP-9
-
Lalu MM, Gao CQ, Schulz R. (2003) Matrix metalloproteinase inhibitors attenuate endotoxemia induced cardiac dysfunction: a potential role for MMP-9. Mol. Cell. Biochem. 251:61-6.
-
(2003)
Mol. Cell. Biochem
, vol.251
, pp. 61-66
-
-
Lalu, M.M.1
Gao, C.Q.2
Schulz, R.3
-
105
-
-
30444460117
-
Roles of MMP-2/-9 in cardiac dysfunction during early multiple organ failure in an ovine animal model
-
Wohlschlaeger J, et al. (2005) Roles of MMP-2/-9 in cardiac dysfunction during early multiple organ failure in an ovine animal model. Pathol. Res. Pract. 201:809-17.
-
(2005)
Pathol. Res. Pract
, vol.201
, pp. 809-817
-
-
Wohlschlaeger, J.1
-
106
-
-
0034730306
-
iNOS gene expression modulates microvascular responsiveness in endotoxin-challenged mice
-
Boyle WA 3rd, et al. (2000) iNOS gene expression modulates microvascular responsiveness in endotoxin-challenged mice. Circ. Res. 87:E18-24.
-
(2000)
Circ. Res
, vol.87
-
-
Boyle 3rd, W.A.1
-
107
-
-
0036100141
-
Myocardial dysfunction in sepsis: No role for
-
Belcher E, Mitchell J, Evans T. (2002) Myocardial dysfunction in sepsis: no role for NO? Heart 87:507-9.
-
(2002)
Heart
, vol.87
, pp. 507-509
-
-
Belcher, E.1
Mitchell, J.2
Evans, T.3
-
108
-
-
0036897624
-
Nitric oxide and myocardial function in heart failure: Friend or foe?
-
Cotton JM, Kearney MT, Shah AM. (2002) Nitric oxide and myocardial function in heart failure: friend or foe? Heart 88:564-6.
-
(2002)
Heart
, vol.88
, pp. 564-566
-
-
Cotton, J.M.1
Kearney, M.T.2
Shah, A.M.3
-
109
-
-
0032891779
-
Role of nitric oxide and cGMP in human septic serum-induced depression of cardiac myocyte contractility
-
Kumar A, et al. (1999) Role of nitric oxide and cGMP in human septic serum-induced depression of cardiac myocyte contractility. Am. J. Physiol. 276:R265-76.
-
(1999)
Am. J. Physiol
, vol.276
-
-
Kumar, A.1
-
110
-
-
0028288683
-
Acute effects of nitric oxide on left ventricular relaxation and diastolic distensibility in humans: Assessment by bicoronary sodium nitroprusside infusion
-
Paulus WJ, Vantrimpont PJ, Shah AM. (1994) Acute effects of nitric oxide on left ventricular relaxation and diastolic distensibility in humans: assessment by bicoronary sodium nitroprusside infusion. Circulation 89:2070-8.
-
(1994)
Circulation
, vol.89
, pp. 2070-2078
-
-
Paulus, W.J.1
Vantrimpont, P.J.2
Shah, A.M.3
-
111
-
-
0034687080
-
Congenital deficiency of nitric oxide synthase 2 protects against endotoxininduced myocardial dysfunction in mice
-
Ullrich R, et al. (2000) Congenital deficiency of nitric oxide synthase 2 protects against endotoxininduced myocardial dysfunction in mice. Circulation 102:1440-6.
-
(2000)
Circulation
, vol.102
, pp. 1440-1446
-
-
Ullrich, R.1
-
112
-
-
0344872651
-
A selective inducible NOS dimerization inhibitor prevents systemic, cardiac, and pulmonary hemodynamic dysfunction in endotoxemic mice
-
Ichinose F, et al. (2003) A selective inducible NOS dimerization inhibitor prevents systemic, cardiac, and pulmonary hemodynamic dysfunction in endotoxemic mice. Am. J. Physiol. Heart Circ. Physiol. 285:H2524-30.
-
(2003)
Am. J. Physiol. Heart Circ. Physiol
, vol.285
-
-
Ichinose, F.1
-
113
-
-
31344453460
-
Role of inducible nitric oxide synthase in the reduced responsiveness of the myocardium to catecholamines in a hyperdynamic, murine model of septic shock
-
Barth E, et al. (2006) Role of inducible nitric oxide synthase in the reduced responsiveness of the myocardium to catecholamines in a hyperdynamic, murine model of septic shock. Crit. Care Med. 34:307-13.
-
(2006)
Crit. Care Med
, vol.34
, pp. 307-313
-
-
Barth, E.1
-
114
-
-
0032990946
-
An open-label dose escalation study of the nitric oxide synthase inhibitor, N(G)-methyl-L-arginine hydrochloride (546C88), in patients with septic shock. Glaxo Wellcome International Septic Shock Study Group
-
Grover R, et al. (1999) An open-label dose escalation study of the nitric oxide synthase inhibitor, N(G)-methyl-L-arginine hydrochloride (546C88), in patients with septic shock. Glaxo Wellcome International Septic Shock Study Group. Crit. Care Med. 27:913-22.
-
(1999)
Crit. Care Med
, vol.27
, pp. 913-922
-
-
Grover, R.1
-
115
-
-
0029919366
-
Peroxynitrite-induced cardiac myocyte injury
-
Ishida H, Ichimori K, Hirota Y, Fukahori M, Nakazawa H. (1996) Peroxynitrite-induced cardiac myocyte injury. Free Radic. Biol. Med. 20:343-50.
-
(1996)
Free Radic. Biol. Med
, vol.20
, pp. 343-350
-
-
Ishida, H.1
Ichimori, K.2
Hirota, Y.3
Fukahori, M.4
Nakazawa, H.5
-
116
-
-
0032482126
-
Inhibition of rat cardiac muscle contraction and mitochondrial respiration by endogenous peroxynitrite formation during posthypoxic reoxygenation
-
Xie YW, Kaminski PM, Wolin MS. (1998) Inhibition of rat cardiac muscle contraction and mitochondrial respiration by endogenous peroxynitrite formation during posthypoxic reoxygenation. Circ. Res. 82:891-7.
-
(1998)
Circ. Res
, vol.82
, pp. 891-897
-
-
Xie, Y.W.1
Kaminski, P.M.2
Wolin, M.S.3
-
117
-
-
2942601560
-
Peroxynitrite decomposition catalysts prevent myocardial dysfunction and inflammation in endotoxemic rats
-
Lancel S, et al. (2004) Peroxynitrite decomposition catalysts prevent myocardial dysfunction and inflammation in endotoxemic rats. J. Am. Coll. Cardiol. 43:2348-58.
-
(2004)
J. Am. Coll. Cardiol
, vol.43
, pp. 2348-2358
-
-
Lancel, S.1
-
118
-
-
10644281080
-
Mitochondrial dysfunction in septic shock and multiple organ dysfunction syndrome
-
Crouser ED. (2004) Mitochondrial dysfunction in septic shock and multiple organ dysfunction syndrome. Mitochondrion 4:729-41.
-
(2004)
Mitochondrion
, vol.4
, pp. 729-741
-
-
Crouser, E.D.1
-
119
-
-
0036487275
-
Mitochondrial dysfunction in sepsis: Evidence from bacteraemic baboons and endotoxaemic rabbits
-
Gellerich FN, et al. (2002) Mitochondrial dysfunction in sepsis: evidence from bacteraemic baboons and endotoxaemic rabbits. Biosci. Rep. 22:99-113.
-
(2002)
Biosci. Rep
, vol.22
, pp. 99-113
-
-
Gellerich, F.N.1
-
120
-
-
0037142987
-
Association between mitochondrial dysfunction and severity and outcome of septic shock
-
Brealey D, et al. (2002) Association between mitochondrial dysfunction and severity and outcome of septic shock. Lancet 360:219-23.
-
(2002)
Lancet
, vol.360
, pp. 219-223
-
-
Brealey, D.1
-
121
-
-
1342331785
-
Mitochondrial dysfunction in a long-term rodent model of sepsis and organ failure
-
Brealey D, et al. (2004) Mitochondrial dysfunction in a long-term rodent model of sepsis and organ failure. Am. J. Physiol. Regul. Integr. Comp. Physiol. 286:R491-7.
-
(2004)
Am. J. Physiol. Regul. Integr. Comp. Physiol
, vol.286
-
-
Brealey, D.1
-
122
-
-
0036174522
-
Endotoxin-induced mitochondrial damage correlates with impaired respiratory activity
-
Crouser ED, Julian MW, Blaho DV, Pfeiffer DR. (2002) Endotoxin-induced mitochondrial damage correlates with impaired respiratory activity. Crit. Care Med. 30:276-84.
-
(2002)
Crit. Care Med
, vol.30
, pp. 276-284
-
-
Crouser, E.D.1
Julian, M.W.2
Blaho, D.V.3
Pfeiffer, D.R.4
-
123
-
-
1242275126
-
Abnormal permeability of inner and outer mitochondrial membranes contributes independently to mitochondrial dysfunction in the liver during acute endotoxemia
-
Crouser ED, et al. (2004) Abnormal permeability of inner and outer mitochondrial membranes contributes independently to mitochondrial dysfunction in the liver during acute endotoxemia. Crit. Care Med. 32:478-88.
-
(2004)
Crit. Care Med
, vol.32
, pp. 478-488
-
-
Crouser, E.D.1
-
124
-
-
5444246214
-
Lipopolysaccharide induces oxidative cardiac mitochondrial damage and biogenesis
-
Suliman HB, Welty-Wolf KE, Carraway M, Tatro L, Piantadosi CA. (2004) Lipopolysaccharide induces oxidative cardiac mitochondrial damage and biogenesis. Cardiovasc. Res. 64:279-88.
-
(2004)
Cardiovasc. Res
, vol.64
, pp. 279-288
-
-
Suliman, H.B.1
Welty-Wolf, K.E.2
Carraway, M.3
Tatro, L.4
Piantadosi, C.A.5
-
125
-
-
0345743533
-
Metabolic dysfunction and depletion of mitochondria in hearts of septic rats
-
Watts JA, Kline JA, Thornton LR, Grattan RM, Brar SS. (2004) Metabolic dysfunction and depletion of mitochondria in hearts of septic rats. J. Mol. Cell Cardiol. 36:141-50.
-
(2004)
J. Mol. Cell Cardiol
, vol.36
, pp. 141-150
-
-
Watts, J.A.1
Kline, J.A.2
Thornton, L.R.3
Grattan, R.M.4
Brar, S.S.5
-
126
-
-
33645805025
-
Potential role of poly(adenosine 5′-diphosphate-ribose) polymerase activation in the pathogenesis of myocardial contractile dysfunction associated with human septic shock
-
Soriano FG, et al. (2006) Potential role of poly(adenosine 5′-diphosphate-ribose) polymerase activation in the pathogenesis of myocardial contractile dysfunction associated with human septic shock. Crit. Care Med. 34:1073-9.
-
(2006)
Crit. Care Med
, vol.34
, pp. 1073-1079
-
-
Soriano, F.G.1
-
127
-
-
4644282833
-
Competitive and noncompetitive inhibition of myocardial cytochrome C oxidase in sepsis
-
Levy RJ, Vijayasarathy C, Raj NR, Avadhani NG, Deutschman CS. (2004) Competitive and noncompetitive inhibition of myocardial cytochrome C oxidase in sepsis. Shock 21:110-4.
-
(2004)
Shock
, vol.21
, pp. 110-114
-
-
Levy, R.J.1
Vijayasarathy, C.2
Raj, N.R.3
Avadhani, N.G.4
Deutschman, C.S.5
-
128
-
-
0033126367
-
Impaired energy metabolism in hearts of septic baboons: Diminished activities of Complex I and Complex II of the mitochondrial respiratory chain
-
Gellerich FN, et al. (1999) Impaired energy metabolism in hearts of septic baboons: diminished activities of Complex I and Complex II of the mitochondrial respiratory chain. Shock 11:336-41.
-
(1999)
Shock
, vol.11
, pp. 336-341
-
-
Gellerich, F.N.1
-
129
-
-
0035083570
-
Different sensitivity of rabbit heart and skeletal muscle to endotoxin-induced impairment of mitochondrial function
-
Trumbeckaite S, Opalka JR, Neuhof C, Zierz S, Gellerich FN. (2001) Different sensitivity of rabbit heart and skeletal muscle to endotoxin-induced impairment of mitochondrial function. Eur. J. Biochem. 268:1422-9.
-
(2001)
Eur. J. Biochem
, vol.268
, pp. 1422-1429
-
-
Trumbeckaite, S.1
Opalka, J.R.2
Neuhof, C.3
Zierz, S.4
Gellerich, F.N.5
-
130
-
-
0028870087
-
Reactive oxygen species produced by liver mitochondria of rats in sepsis
-
Taylor DE, Ghio AJ, Piantadosi CA. (1995) Reactive oxygen species produced by liver mitochondria of rats in sepsis. Arch. Biochem. Biophys. 316:70-6.
-
(1995)
Arch. Biochem. Biophys
, vol.316
, pp. 70-76
-
-
Taylor, D.E.1
Ghio, A.J.2
Piantadosi, C.A.3
-
131
-
-
34250733466
-
Mitochondrial dysfunction, bioenergetic impairment, and metabolic down-regulation in sepsis
-
Levy RJ. (2007) Mitochondrial dysfunction, bioenergetic impairment, and metabolic down-regulation in sepsis. Shock 28:24-8.
-
(2007)
Shock
, vol.28
, pp. 24-28
-
-
Levy, R.J.1
-
132
-
-
0036892452
-
Bench-to-bedside review: Cytopathic hypoxia
-
Fink MP. (2002) Bench-to-bedside review: cytopathic hypoxia. Crit. Care 6:491-9.
-
(2002)
Crit. Care
, vol.6
, pp. 491-499
-
-
Fink, M.P.1
-
133
-
-
0036121121
-
Cytopathic hypoxia: Is oxygen use impaired in sepsis as a result of an acquired intrinsic derangement in cellular respiration?
-
Fink MP. (2002) Cytopathic hypoxia: is oxygen use impaired in sepsis as a result of an acquired intrinsic derangement in cellular respiration? Crit. Care Clin. 18:165-75.
-
(2002)
Crit. Care Clin
, vol.18
, pp. 165-175
-
-
Fink, M.P.1
-
135
-
-
33745685397
-
Inhibition of mitochondrial permeability transition prevents sepsis-induced myocardial dysfunction and mortality
-
Larche J, et al. (2006) Inhibition of mitochondrial permeability transition prevents sepsis-induced myocardial dysfunction and mortality. J. Am. Coll. Cardiol. 48:377-85.
-
(2006)
J. Am. Coll. Cardiol
, vol.48
, pp. 377-385
-
-
Larche, J.1
-
136
-
-
0034957413
-
Effect of NF-kappa B inhibition on TNF-alpha-induced apoptosis and downstream pathways in cardiomyocytes
-
Bergmann MW, Loser P, Dietz R, von Harsdorf R. (2001) Effect of NF-kappa B inhibition on TNF-alpha-induced apoptosis and downstream pathways in cardiomyocytes. J. Mol. Cell Cardiol. 33:1223-32.
-
(2001)
J. Mol. Cell Cardiol
, vol.33
, pp. 1223-1232
-
-
Bergmann, M.W.1
Loser, P.2
Dietz, R.3
von Harsdorf, R.4
-
137
-
-
0035008514
-
Differential effects of caspase inhibitors on endotoxin-induced myocardial dysfunction and heart apoptosis
-
Fauvel H, Marchetti P, Chopin C, Formstecher P, Neviere R. (2001) Differential effects of caspase inhibitors on endotoxin-induced myocardial dysfunction and heart apoptosis. Am. J. Physiol. Heart Circ. Physiol. 280:H1608-14.
-
(2001)
Am. J. Physiol. Heart Circ. Physiol
, vol.280
-
-
Fauvel, H.1
Marchetti, P.2
Chopin, C.3
Formstecher, P.4
Neviere, R.5
-
139
-
-
33845453393
-
Antioxidant vitamin therapy alters sepsis-related apoptotic myocardial activity and inflammatory responses
-
Carlson D, Maass DL, White DJ, Tan J, Horton JW. (2006) Antioxidant vitamin therapy alters sepsis-related apoptotic myocardial activity and inflammatory responses. Am. J. Physiol. Heart Circ. Physiol. 291:H2779-89.
-
(2006)
Am. J. Physiol. Heart Circ. Physiol
, vol.291
-
-
Carlson, D.1
Maass, D.L.2
White, D.J.3
Tan, J.4
Horton, J.W.5
-
140
-
-
44649144836
-
-
Buerke U, et al. (2007) Apoptosis contributes to septic cardiomyopathy and is improved by simvastatin therapy. Shock. Aug 2;Publish Ahead of Print.
-
Buerke U, et al. (2007) Apoptosis contributes to septic cardiomyopathy and is improved by simvastatin therapy. Shock. Aug 2;Publish Ahead of Print.
-
-
-
-
141
-
-
30044450067
-
Human serum from patients with septic shock activates transcription factors STAT1, IRF1, and NF-kappaB and induces apoptosis in human cardiac myocytes
-
Kumar A, et al. (2005) Human serum from patients with septic shock activates transcription factors STAT1, IRF1, and NF-kappaB and induces apoptosis in human cardiac myocytes. J. Biol. Chem. 280:42619-26.
-
(2005)
J. Biol. Chem
, vol.280
, pp. 42619-42626
-
-
Kumar, A.1
-
142
-
-
19644369587
-
Ventricular myocyte caspases are directly responsible for endotoxin-induced cardiac dysfunction
-
Lancel S, et al. (2005) Ventricular myocyte caspases are directly responsible for endotoxin-induced cardiac dysfunction. Circulation 111: 2596-604.
-
(2005)
Circulation
, vol.111
, pp. 2596-2604
-
-
Lancel, S.1
-
143
-
-
0035154684
-
Caspase inhibition prevents cardiac dysfunction and heart apoptosis in a rat model of sepsis
-
Neviere R, Fauvel H, Chopin C, Formstecher P, Marchetti P. (2001) Caspase inhibition prevents cardiac dysfunction and heart apoptosis in a rat model of sepsis. Am. J. Respir. Crit. Care Med. 163:218-25.
-
(2001)
Am. J. Respir. Crit. Care Med
, vol.163
, pp. 218-225
-
-
Neviere, R.1
Fauvel, H.2
Chopin, C.3
Formstecher, P.4
Marchetti, P.5
-
144
-
-
0037084168
-
Protective effects of cyclosporin A from endotoxin-induced myocardial dysfunction and apoptosis in rats
-
Fauvel H, et al. (2002) Protective effects of cyclosporin A from endotoxin-induced myocardial dysfunction and apoptosis in rats. Am. J. Respir. Crit. Care Med. 165:449-55.
-
(2002)
Am. J. Respir. Crit. Care Med
, vol.165
, pp. 449-455
-
-
Fauvel, H.1
-
145
-
-
14944354292
-
Expression of apoptosis regulatory factors during myocardial dysfunction in endotoxemic rats
-
Lancel S, et al. (2005) Expression of apoptosis regulatory factors during myocardial dysfunction in endotoxemic rats. Crit. Care Med. 33:492-6.
-
(2005)
Crit. Care Med
, vol.33
, pp. 492-496
-
-
Lancel, S.1
-
146
-
-
0034503532
-
The biology of myocardial hibernation
-
Heusch G, Schulz R. (2000) The biology of myocardial hibernation. Trends Cardiovasc. Med. 10: 108-14.
-
(2000)
Trends Cardiovasc. Med
, vol.10
, pp. 108-114
-
-
Heusch, G.1
Schulz, R.2
-
147
-
-
0029865769
-
Calcium responsiveness in regional myocardial short-term hibernation and stunning in the in situ porcine heart: Inotropic responses to postextrasystolic potentiation and intracoronary calcium
-
Heusch G, Rose J, Skyschally A, Post H, Schulz R. (1996) Calcium responsiveness in regional myocardial short-term hibernation and stunning in the in situ porcine heart: inotropic responses to postextrasystolic potentiation and intracoronary calcium. Circulation 93:1556-66.
-
(1996)
Circulation
, vol.93
, pp. 1556-1566
-
-
Heusch, G.1
Rose, J.2
Skyschally, A.3
Post, H.4
Schulz, R.5
-
148
-
-
0037007057
-
Myocardial hibernation: Restorative or preterminal sleep?
-
Sawyer DB, Loscalzo J. (2002) Myocardial hibernation: restorative or preterminal sleep? Circulation 105:1517-9.
-
(2002)
Circulation
, vol.105
, pp. 1517-1519
-
-
Sawyer, D.B.1
Loscalzo, J.2
-
149
-
-
0030893394
-
Chronic myocardial hibernation in humans: From bedside to bench
-
Vanoverschelde JL, et al. (1997) Chronic myocardial hibernation in humans: from bedside to bench. Circulation 95:1961-71.
-
(1997)
Circulation
, vol.95
, pp. 1961-1971
-
-
Vanoverschelde, J.L.1
-
150
-
-
0342506453
-
Hibernating myocardium: An incomplete adaptation to ischemia
-
Elsasser A, et al. (1997) Hibernating myocardium: an incomplete adaptation to ischemia. Circulation 96:2920-31.
-
(1997)
Circulation
, vol.96
, pp. 2920-2931
-
-
Elsasser, A.1
-
151
-
-
2942543198
-
Human hibernating myocardium is jeopardized by apoptotic and autophagic cell death
-
Elsasser A, et al. (2004) Human hibernating myocardium is jeopardized by apoptotic and autophagic cell death. J. Am. Coll. Cardiol. 43:2191-9.
-
(2004)
J. Am. Coll. Cardiol
, vol.43
, pp. 2191-2199
-
-
Elsasser, A.1
-
152
-
-
0037007058
-
Increased myocardial gene expression of tumor necrosis factor-alpha and nitric oxide synthase-2: A potential mechanism for depressed myocardial function in hibernating myocardium in humans
-
Kalra DK, et al. (2002) Increased myocardial gene expression of tumor necrosis factor-alpha and nitric oxide synthase-2: a potential mechanism for depressed myocardial function in hibernating myocardium in humans. Circulation 105:1537-40.
-
(2002)
Circulation
, vol.105
, pp. 1537-1540
-
-
Kalra, D.K.1
|