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Volumn 102, Issue 12, 2000, Pages 1440-1446

Congenital deficiency of nitric oxide synthase 2 protects against endotoxin-induced myocardial dysfunction in mice

Author keywords

Echocardiography; Heart failure; Inflammation; Inhibitors; Sepsis

Indexed keywords

CYCLIC GMP; ENDOTOXIN; NITRIC OXIDE SYNTHASE;

EID: 0034687080     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.102.12.1440     Document Type: Article
Times cited : (114)

References (27)
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  • 20
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    • N-ω-Amino-L-arginine, an inhibitor of nitric oxide synthase, raises vascular resistance but increases mortality rates in awake canines challenged with endotoxin
    • (1992) J Exp Med , vol.176 , pp. 1175-1182
    • Cobb, J.P.1    Natanson, C.2    Hoffman, W.D.3
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    • Echocardiographic and survival studies in mice undergoing endotoxic shock: Effects of genetic ablation of inducible nitric oxide synthase and pharmacologic antagonism of platelet-activating factor
    • (1999) J Surg Res , vol.86 , pp. 198-205
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.