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Volumn 104, Issue 25, 2001, Pages 3137-3144

Activation of cardiac endothelium as a compensatory component in endotoxin-induced cardiomyopathy: Role of endothelin, prostaglandins, and nitric oxide

Author keywords

Endocardium; Infection; Myocardium; Nitric oxide synthase; Prostaglandins

Indexed keywords

3 NITROTYROSINE; BACTERIUM LIPOPOLYSACCHARIDE; CYCLO(DEXTRO TRYPTOPHYL DEXTRO ASPARTYLPROLYL DEXTRO VALYLLEUCYL); CYCLOOXYGENASE 2; ENDOTHELIN; ENDOTHELIN RECEPTOR ANTAGONIST; ENDOTOXIN; INDOMETACIN; ISOENZYME; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; PEROXYNITRITE; PROSTAGLANDIN; SODIUM CHLORIDE;

EID: 0035909920     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/hc5001.100662     Document Type: Article
Times cited : (42)

References (29)
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  • 24
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    • Endotoxin-induced myocardial tumor necrosis factor-α synthesis depresses contractility of isolated rat hearts: Evidence for a role of sphingosine and cyclooxygenase -2-derived thromboxane production
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    • Grandel, U.1    Fink, L.2    Blum, A.3
  • 27
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    • Nitric oxide synthase is not involved in cardiac contractile dysfunction in a rat model of endotoxemia without shock
    • (1997) Shock , vol.7 , pp. 111-118
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  • 28
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.