Bax deficiency promotes an up-regulation of BimEL and Bak during striatal and cortical postnatal development, and after excitotoxic injury

Molecular and Cellular Neuroscience

Volumn 37, Issue 4, 2008, Pages 663-672

  Gavalda N ^a   Perez Navarro E ^a   Garcia Martinez J M ^a   Marco, S ^a   Benito, A ^b   Alberch, J ^b  

^a UNIVERSITY OF BARCELONA   (Spain)

^b HOSPITAL UNIVERSITARIO MARQUÉS DE VALDECILLA   (Spain)

Author keywords

Apoptosis; Basal ganglia; Bcl 2, BH 3 only proteins; Knockout

Indexed keywords

BIM PROTEIN; CASPASE 3; CYTOCHROME C; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BRAIN DEVELOPMENT; CELL DEATH; CELL FRACTIONATION; CONTROLLED STUDY; ENZYME ACTIVATION; EXCITOTOXICITY; FEMALE; GENOTYPE; MALE; MITOCHONDRION; MOUSE; NONHUMAN; POSTNATAL DEVELOPMENT; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN SECRETION;

ANIMALS; ANIMALS, NEWBORN; APOPTOSIS; APOPTOSIS REGULATORY PROTEINS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; CELL DEATH; CEREBRAL CORTEX; CORPUS STRIATUM; FEMALE; MALE; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; PROTO-ONCOGENE PROTEINS; QUINOLINIC ACID; UP-REGULATION;

ANIMALIA; MUS;

EID: 41349083731     PISSN: 10447431     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mcn.2007.12.013     Document Type: Article

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