γ-Cleavage-Independent Functions of Presenilin, Nicastrin, and Aph-1 Regulate Cell-Junction Organization and Prevent Tau Toxicity In Vivo (DOI:10.1016/j.neuron.2006.03.038);γ-Cleavage-Independent Functions of Presenilin, Nicastrin, and Aph-1 Regulate Cell-Junction Organization and Prevent Tau Toxicity In Vivo

Neuron

Volumn 50, Issue 3, 2006, Pages 359-375

  Doglio, Laura E ^a,b   Kanwar, Ritu ^c   Jackson, George R ^d   Perez, Mar ^e   Avila, Jesús ^e   Dingwall, Colin ^f   Dotti, Carlos G ^a,b   Fortini, Mark E ^c   Feiguin, Fabián ^a  

^a UNIVERSITY OF TURIN   (Italy)

^b DEPARTMENT OF PLANT SYSTEMS BIOLOGY   (Belgium)

^c NATIONAL CANCER INSTITUTE   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

^e UNIVERSIDAD AUTÓNOMA DE MADRID   (Spain)

^f GLAXOSMITHKLINE   (United Kingdom)

Author keywords

HUMDISEASE; MOLNEURO; SYSNEURO

Indexed keywords

ANTERIOR PHARYNX DEFECTIVE 1 PROTEIN; GAMMA SECRETASE; GLYCOGEN SYNTHASE KINASE; MEMBRANE PROTEIN; NICASTRIN; PHOSPHATIDYLINOSITOL 3 KINASE; PRESENILIN 1; PROTEIN KINASE B; TAU PROTEIN;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL JUNCTION; CELLULAR DISTRIBUTION; DROSOPHILA; ENZYME ACTIVITY; ENZYME INHIBITION; IN VIVO STUDY; NERVE DEGENERATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; REGULATORY MECHANISM; SIGNAL TRANSDUCTION;

EID: 33646149630     PISSN: 08966273     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuron.2007.01.012     Document Type: Erratum

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