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Volumn 1024, Issue , 2004, Pages 54-71
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Physiological and pathological consequences of the interactions of the p53 tumor suppressor with the glucocorticoid, androgen, and estrogen receptors
a
b
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Author keywords
Addison's disease; Anemia; Cancer progression; Cushing's syndrome; Hematological malignancies; Skin carcinogenesis; Stress erythropoiesis
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Indexed keywords
ANDROGEN;
ANDROGEN RECEPTOR;
CCAAT ENHANCER BINDING PROTEIN;
DEXAMETHASONE;
ESTROGEN RECEPTOR;
GENE PRODUCT;
GLUCOCORTICOID;
GLUCOCORTICOID RECEPTOR;
GLUCOSE;
ONCOPROTEIN;
PROTEIN BAX;
PROTEIN MDM2;
PROTEIN P21;
PROTEIN P53;
UBIQUITIN PROTEIN LIGASE;
APOPTOSIS;
AUTOIMMUNE DISEASE;
BREAST CARCINOMA;
CARCINOGENESIS;
CELL PROLIFERATION;
COLON ADENOCARCINOMA;
CONFERENCE PAPER;
DIMERIZATION;
DNA BINDING;
DNA DAMAGE;
ERYTHROPOIESIS;
GENE ACTIVATION;
GENE DOSAGE;
GENE EXPRESSION;
GENE INACTIVATION;
GENE INTERACTION;
GENE OVEREXPRESSION;
GENE THERAPY;
GENETIC TRANSCRIPTION;
GLUCOSE METABOLISM;
HOMEOSTASIS;
HUMAN;
HYPOXIA;
INFLAMMATION;
LEUKEMIA;
LYMPHOMA;
NEUROBLASTOMA;
ONCOGENE C MYC;
PATHOLOGY;
PHEOCHROMOCYTOMA;
PHOSPHORYLATION;
PHYSIOLOGY;
PROTEIN PROTEIN INTERACTION;
PROTEIN STABILITY;
REPRODUCTION;
SKIN CARCINOGENESIS;
THYMOCYTE;
TRANSCRIPTION REGULATION;
TUMOR SUPPRESSOR GENE;
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EID: 3042795223
PISSN: 00778923
EISSN: None
Source Type: Book Series
DOI: 10.1196/annals.1321.005 Document Type: Conference Paper |
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Times cited : (72)
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References (137)
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