Ionizing radiation-dependent γ-H2AX focus formation requires ataxia telangiectasia mutated and ataxia telangiectasia mutated and Rad3-related

Molecular Biology of the Cell

Volumn 16, Issue 5, 2005, Pages 2566-2576

  Friesner, Joanna D ^a   Liu, Bo ^a   Culligan, Kevin ^a   Britt, Anne B ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ATM PROTEIN; ATR PROTEIN; DOUBLE STRANDED DNA; GAMMA HISTONE H2AX; HISTONE H2A; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE C; UNCLASSIFIED DRUG;

ARABIDOPSIS; ARTICLE; CELL CYCLE; CELL CYLE M PHASE; CONTROLLED STUDY; DNA DAMAGE; DNA REPAIR; DNA REPLICATION; HUMAN; HUMAN CELL; IONIZING RADIATION; MAMMAL; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION; RADIOSENSITIVITY; YEAST;

ARABIDOPSIS; ARABIDOPSIS PROTEINS; CELL CYCLE PROTEINS; CELL DIVISION; CELL LINE; DNA REPAIR; DNA REPLICATION; GENES, PLANT; HISTONES; HUMANS; MUTATION; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; RADIATION TOLERANCE; SPECIES SPECIFICITY;

ARABIDOPSIS; ATAXIA; ATAXIA TELANGIECTASIA; MAMMALIA;

EID: 18244385198     PISSN: 10591524     EISSN: None     Source Type: Journal    
DOI: 10.1091/mbc.E04-10-0890     Document Type: Article

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