Early and selective loss of neuromuscular synapse subtypes with low sprouting competence in motoneuron diseases

Journal of Neuroscience

Volumn 20, Issue 7, 2000, Pages 2534-2542

  Frey, Dunja ^a   Schneider, Corinna ^a   Xu, Lan ^a   Borg, Jacques ^b   Spooren, Will ^c   Caroni, Pico ^a  

^a FRIEDRICH MIESCHER INSTITUTE FOR BIOMEDICAL RESEARCH   (Switzerland)

^b UNIVERSITÉ DE STRASBOURG   (France)

^c NOVARTIS PHARMA AG   (Switzerland)

Author keywords

Anatomical plasticity; Botulinum toxin A; GAP43; Motoneuron disease; Neuromuscular junction; Selective vulnerability; Synapse subtypes

Indexed keywords

BOTULINUM TOXIN A; NEUROMODULIN;

AGING; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DEGENERATIVE DISEASE; DENERVATION; DISEASE COURSE; GENE OVEREXPRESSION; HISTOLOGY; IMMUNOCYTOCHEMISTRY; MOTOR NEURON DISEASE; MOUSE; MUSCLE FATIGUE; NERVE CELL PLASTICITY; NEUROMUSCULAR SYNAPSE; NONHUMAN; PRIORITY JOURNAL; SYNAPTIC TRANSMISSION; TRICEPS SURAE MUSCLE;

EID: 0034175513     PISSN: 02706474     EISSN: None     Source Type: Journal    
DOI: 10.1523/jneurosci.20-07-02534.2000     Document Type: Article

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