The Bax subfamily of Bcl2-related proteins is essential for apoptotic signal transduction by c-Jun NH2-terminal kinase

Molecular and Cellular Biology

Volumn 22, Issue 13, 2002, Pages 4929-4942

  Lei, Kui ^a   Nimnual, Anjaruwee ^a   Zong, Wei Xing ^a   Kennedy, Norman J ^a   Flavell, Richard A ^a   Thompson, Craig B ^a   Bar Sagi, Dafna ^a   Davis, Roger J ^a  

^a UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE; CASPASE 3; CYTOCHROME C; MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2; STRESS ACTIVATED PROTEIN KINASE; TRANSCRIPTION FACTOR AP 1;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DEATH; CONTROLLED STUDY; GENE DISRUPTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; STRESS;

ANIMALS; APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; CASPASE 3; CASPASES; CELLS, CULTURED; CRICETINAE; CYTOCHROME C GROUP; ENZYME ACTIVATION; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MAP KINASE KINASE 7; MEMBRANE PROTEINS; MICE; MITOCHONDRIA; MITOGEN-ACTIVATED PROTEIN KINASE KINASES; MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; RECOMBINANT PROTEINS; SIGNAL TRANSDUCTION; TRANSCRIPTION FACTOR AP-1; ULTRAVIOLET RAYS;

ANIMALIA;

EID: 0036269755     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.22.13.4929-4942.2002     Document Type: Article

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