Cross-talk between pro-inflammatory transcription factors and glucocorticoids

Immunology and Cell Biology

Volumn 79, Issue 4, 2001, Pages 376-384

  Adcock, I M ^a   Caramori, G ^a  

^a NATIONAL HEART AND LUNG INSTITUTE   (United Kingdom)

Author keywords

Acetylation; AP 1; Asthma; Glucocorticoid; Histones; Inflammation; NF B; Receptors; Transcription factor

Indexed keywords

CHEMOKINE; CYTOKINE; DEXAMETHASONE; EOTAXIN; GLUCOCORTICOID; GRANULOCYTE MACROPHAGE COLONY STIMULATING FACTOR; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 1BETA; STRESS ACTIVATED PROTEIN KINASE; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR AP 1;

ARTICLE; ASTHMA; CYTOKINE PRODUCTION; DRUG EFFECT; DRUG MECHANISM; GENE INDUCTION; GENE REPRESSION; GENETIC TRANSCRIPTION; HEALTH CARE COST; HUMAN; INFLAMMATION; MEDIATOR RELEASE;

ACETYLATION; ANTI-ASTHMATIC AGENTS; ANTI-INFLAMMATORY AGENTS; ASTHMA; CYTOKINES; GENE EXPRESSION REGULATION; GLUCOCORTICOIDS; HISTONES; HUMANS; INFLAMMATION MEDIATORS; MODELS, BIOLOGICAL; RECEPTORS, GLUCOCORTICOID; SIGNAL TRANSDUCTION; STEROIDS; TRANSCRIPTION FACTORS;

EID: 0034889129     PISSN: 08189641     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1440-1711.2001.01025.x     Document Type: Article

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