The senescent bystander effect is caused by ROS-activated NF-κB signalling

Mechanisms of Ageing and Development

Volumn 170, Issue , 2018, Pages 30-36

  Nelson, Glyn ^a,b   Kucheryavenko, Olena ^a,c   Wordsworth, James ^a   von Zglinicki, Thomas ^a  

^a NEWCASTLE UNIVERSITY   (United Kingdom)

^b NEWCASTLE UNIVERSITY   (United Kingdom)

^c FEDERAL INSTITUTE FOR RISK ASSESSMENT   (Germany)

Author keywords

Bystander; Mitochondria; NF B; Reactive oxygen; Senescence

Indexed keywords

CARISOPRODOL; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 6; INTERLEUKIN 8; MITOGEN ACTIVATED PROTEIN KINASE P38; REACTIVE OXYGEN METABOLITE; IL6 PROTEIN, HUMAN; IL8 PROTEIN, HUMAN;

AGING; ARTICLE; BYSTANDER EFFECT; CELL AGING; CELL DIVISION; CELL PROLIFERATION; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DNA DAMAGE; EMBRYO; FIBROBLAST; HUMAN; HUMAN CELL; IMMUNOFLUORESCENCE; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; MITOPHAGY; PRIORITY JOURNAL; PROTEIN LOCALIZATION; SIGNAL TRANSDUCTION; X IRRADIATION; CELL LINE; METABOLISM; PATHOLOGY;

BYSTANDER EFFECT; CELL LINE; CELLULAR SENESCENCE; DNA DAMAGE; FIBROBLASTS; HUMANS; INTERLEUKIN-6; INTERLEUKIN-8; NF-KAPPA B; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION;

EID: 85028344773     PISSN: 00476374     EISSN: 18726216     Source Type: Journal    
DOI: 10.1016/j.mad.2017.08.005     Document Type: Article

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