Absence of the complement anaphylatoxin C3a receptor suppresses Th2 effector functions in a murine model of pulmonary allergy

Journal of Immunology

Volumn 169, Issue 10, 2002, Pages 5926-5933

  Drouin, Scott M ^a   Corry, David B ^a   Hollman, Travis J ^a   Kildsgaard, Jens ^a   Wetsel, Rick A ^a  

^a UNIVERSITY OF TEXAS MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANAPHYLATOXIN; ANTIGEN; COMPLEMENT COMPONENT C3 RECEPTOR; COMPLEMENT COMPONENT C3A; COMPLEMENT COMPONENT C3A RECEPTOR; IMMUNOGLOBULIN E; INTERLEUKIN 13; INTERLEUKIN 4; INTERLEUKIN 5; OVALBUMIN; UNCLASSIFIED DRUG;

AIRWAY; AIRWAY OBSTRUCTION; ANIMAL EXPERIMENT; ANIMAL MODEL; ANTIBODY TITER; ARTICLE; ASPERGILLUS FUMIGATUS; ASTHMA; ATTENUATION; CELL CULTURE; COMPLEMENT ACTIVATION; EOSINOPHIL; INFLAMMATION; KNOCKOUT MOUSE; LUNG; LUNG LAVAGE; MOUSE; MUCUS; NONHUMAN; PLETHYSMOGRAPHY; PRIORITY JOURNAL; PROVOCATION TEST; RESPIRATORY TRACT ALLERGY; TH2 CELL; WILD TYPE;

EID: 0037111427     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.169.10.5926     Document Type: Article

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